Ask your doctor if sleeping pill use even counts as sleeping.
http://onlinelibrary.wiley.com/doi/10.1046/j.1365-2869.2002.00316.x/abstract
Article first published online: 11 DEC 2002
DOI: 10.1046/j.1365-2869.2002.00316.x
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Keywords:
- stroke;
- sleep;
- EEG;
- activity;
- insomnia;
- hypersomnia;
- Epworth sleepiness score;
- outcome
SUMMARY
The
evolution of subjective sleep and sleep electroencephalogram (EEG)
after hemispheric stroke have been rarely studied and the relationship
of sleep variables to stroke outcome is essentially unknown. We studied
27 patients with first hemispheric ischaemic stroke and no sleep apnoea
in the acute (1–8 days), subacute (9–35 days), and chronic phase
(5–24 months) after stroke. Clinical assessment included estimated sleep
time per 24 h (EST) and Epworth sleepiness score (ESS) before stroke,
as well as EST, ESS and clinical outcome after stroke. Sleep EEG data
from stroke patients were compared with data from 11 hospitalized
controls and published norms. Changes in EST (>2 h, 38% of patients)
and ESS (>3 points, 26%) were frequent but correlated poorly with
sleep EEG changes. In the chronic phase no significant differences in
sleep EEG between controls and patients were found. High sleep
efficiency and low wakefulness after sleep onset in the acute phase were
associated with a good long-term outcome. These two sleep EEG variables
improved significantly from the acute to the subacute and chronic
phase. In conclusion, hemispheric strokes can cause insomnia,
hypersomnia or changes in sleep needs but only rarely persisting sleep
EEG abnormalities. High sleep EEG continuity in the acute phase of
stroke heralds a good clinical outcome.
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