Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, December 18, 2013

Interactions of black tea polyphenols with human gut microbiota: implications for gut and cardiovascular health

I couldn't see any references to cardiovascular health but then this is just the abstract. You'll have to ask your doctor  how many cups of black tea you should be drinking immediately post-stroke.
And since I was only mobile in a wheelchair you would need to make sure your wheelchair has cup holders for transporting it back to your room from the cafeteria.
http://ajcn.nutrition.org/content/98/6/1631S.abstract?sid=3a949d69-2700-4665-a8fe-c4f1bb2c9554

  1. Doris M Jacobs
+ Author Affiliations
  1. 1From Unilever Discover Vlaardingen, Vlaardingen, Netherlands (JvD, EEV, FvD, LR, RD, and DMJ); the Laboratory of Biophysics and Wageningen NMR Centre (JvD and JV), and the Laboratory of Biochemistry (JV and JJJvdH), Wageningen University, Wageningen, Netherlands; Plant Research International, Wageningen, Netherlands (VG-R, RdV, and JJJvdH); the Netherlands Metabolomics Centre, Leiden, Netherlands (JvD, FvD, RdV, JV, JJJvdH, and DMJ); and the Centre for Biosystems Genomics, Wageningen, Netherlands (RdV and VG-R).
+ Author Notes
  • 2 Presented at the conference “Fifth International Scientific Symposium on Tea and Human Health,” held at the US Department of Agriculture, Washington, DC, 19 September 2012. The conference was organized by Jeffrey Blumberg, Tufts University, Boston, MA, and a Steering Committee including representatives from each of the symposium cosponsors: the American Cancer Society, the American College of Nutrition, the American Institute for Cancer Research, the American Medical Women's Association, the American Society for Nutrition, and the Linus Pauling Institute. The symposium was underwritten by the Tea Council of the USA. Its contents are solely the responsibility of the authors and do not necessarily represent the official views of the Tea Council of the USA or the cosponsoring organizations.
  • 3 This work was jointly financed by Unilever and the Netherlands Metabolomics Centre (NMC), which is part of the Netherlands Genomics Initiative/Netherlands Organisation for Scientific Research. JvD received travel support from the Tea Council of the USA for speaking at the Fifth International Scientific Symposium on Tea and Human Health.
  • 4 Address correspondence to J van Duynhoven, Unilever Discover Vlaardingen, Olivier van Noortlaan 120, 3133AT, Vlaardingen, Netherlands. E-mail: john-van.duynhoven@unilever.com.

Abstract

Epidemiologic studies have convincingly associated consumption of black tea with reduced cardiovascular risk. Research on the bioactive molecules has traditionally been focused on polyphenols, such as catechins. Black tea polyphenols (BTPs), however, mainly consist of high-molecular-weight species that predominantly persist in the colon. There, they can undergo a wide range of bioconversions by the resident colonic microbiota but can in turn also modulate gut microbial diversity. The impact of BTPs on colon microbial composition can now be assessed by microbiomics technologies. Novel metabolomics platforms coupled to de novo identification are currently available to cover the large diversity of BTP bioconversions by the gut microbiota. Nutrikinetic modeling has been proven to be critical for defining nutritional phenotypes related to gut microbial bioconversion capacity. The bioactivity of circulating metabolites has been studied only to a certain extent. Bioassays dedicated to specific aspects of gut and cardiovascular health have been used, although often at physiologically irrelevant concentrations and with limited coverage of relevant metabolite classes and their conjugated forms. Evidence for cardiovascular benefits of BTPs points toward antiinflammatory and blood pressure–lowering properties and improvement in platelet and endothelial function for specific microbial bioconversion products. Clearly, more work is needed to fill in existing knowledge gaps and to assess the in vitro and in vivo bioactivity of known and newly identified BTP metabolites. It is also of interest to assess how phenotypic variation in gut microbial BTP bioconversion capacity relates to gut and cardiovascular health predisposition.

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