Deans' stroke musings

Changing stroke rehab and research worldwide now.Time is Brain!Just think of all the trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 493 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It's quite disgusting that this information is not available from every stroke association and doctors group.
My back ground story is here:

Sunday, January 29, 2017

Depression confers risk for CVD in similar magnitude to obesity, high cholesterol in men

Does your doctor know about the correct way to treat your stroke depression? Maybe by not having you so fucking disabled because you are being treated for the  5 causes of neuronal cascade of death in the first week. Thus leaving you much less disabled and possibly getting to 100% recovery. Treat the cause of the depression not the depression itself. But if your doctor knows nothing about cascade of death prevention I guess you will have to go down the second best route.

Common antidepressant can help stroke patients improve movement and coordination Sept. 2015 

Antidepressants may help people recover from stroke even if they are not depressed Jan. 2013 

Not talk therapy.

Depression confers risk for CVD in similar magnitude to obesity, high cholesterol in men 

According to new data, depressed mood and exhaustion pose as great a CVD risk in men as high cholesterol and obesity.
“There is little doubt that depression is a risk factor for [CVDs],” Karl-Heinz Ladwig, MD, group leader at the Institute of Epidemiology II at the Helmholtz Zentrum München, Neuherberg, Germany, professor of psychosomatic medicine at Technische Universität München’s Klinikum recths der Isar and scientist at Deutsches Zentrum für Herz-Kreislauf-Forschung, Munich, said in a press release. “The question now is: What is the relationship between depression and other risk factors like tobacco smoke, high cholesterol levels, obesity or hypertension — how big a role does each factor play?”
The researchers analyzed 3,428 patients aged 45 to 74 years who participated in one of three MONICA/KORA Augsburg surveys, conducted between 1984 and 1995, to investigate the comparison of the link between traditional somatic risk factors and depressed mood on all-cause mortality prediction and fatal CVD endpoints in an identical-source population.
In 10 years of follow-up covering 31,791 patient-years, 269 fatal CVD events and 557 cases of all-cause mortality were observed.
At baseline, 34% (n = 1,164) of participants experienced depressed mood and exhaustion.
Per 1,000 person-years, the absolute mortality risk for depressed mood and exhaustion was 23.1 cases for all-cause mortality and 11.2 cases for CVD mortality, Ladwig and colleagues wrote.
Depressed mood and exhaustion conferred elevated risk for all-cause and CVD mortality (HR = 1.52; P < .01 for both) at a magnitude higher than hypercholesterolemia and obesity but lower than hypertension, smoking and diabetes, according to the researchers.
Improvement in all-cause and CVD mortality risk prediction from depressed mood and exhaustion was similar to that from hypercholesterolemia and obesity, but substantially lower than that from hypertension, smoking and diabetes, Ladwig and colleagues wrote.
Depressed mood and exhaustion accounted for roughly 15% of the population-attributable risk for all-cause and CVD mortality, placing it around the middle among the classical risk factors, according to the researchers.
“Our data show that depression has a medium effect size within the range of major, non-congenital risk factors for [CVDs],” Ladwig said in the release. “In high-risk patients, the diagnostic investigation of comorbid depression should be standard. This could be registered with simple means.” – by Dave Quaile

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