Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, August 14, 2014

Pontine Microbleeds and Depression in Stroke

This is just so goddamned f*cking simple to identify the problem. Currently our doctors do not give us any objective diagnosis or any proven repeatable way to recover.  With such lack of information depression is inevitable. Stop the neuronal cascade of death and survivors will be much less disabled and less likely to get depressed. Solve the correct problem you blasted idiots.
http://jgp.sagepub.com/content/27/3/159?etoc

  1. W. K. Tang, MD1⇑
  2. X. X. Liu, MPhil1
  3. Y. K. Chen, PhD2
  4. J. Abrigo, MD3
  5. Winnie C. W. Chu, MD3
  6. V. C. T. Mok, MD4
  7. Gabor S. Ungvari, MD, PhD5,6
  8. K. S. Wong, MD4
  1. 1Department of Psychiatry, Chinese University of Hong Kong, Hong Kong SAR, China
  2. 2Department of Neurology, Dongguan People’s Hospital, Dongguan, Guangdong, China
  3. 3Department of Imaging and Interventional Radiology, Chinese University of Hong Kong, Hong Kong SAR, China
  4. 4Department of Medicine and Therapeutics, Chinese University of Hong Kong, Hong Kong SAR, China
  5. 5The University of Notre Dame Australia/Marian Centre, Perth, Australia
  6. 6School of Psychiatry and Clinical Neuroscience, University of Western Australia, Pert, Australia
  1. W. K. Tang, Department of Psychiatry, Shatin Hospital, Shatin, NT, Hong Kong SAR, China. Email: tangwk@cuhk.edu.hk

Abstract

Objectives: Depression is the most common affective disorder following stroke yet the neuroanatomical model of poststroke depression (PSD) remains unclear. This study examined the association between PSD and cerebral microbleeds (CMBs) and hypothesized that CMBs in specific regions would be associated with PSD.
Methods: Of the 4766 patients with first ever or recurrent acute ischemic stroke admitted to the Acute Stroke Unit of the Prince of Wales Hospital between June 2004 and October 2010, 229 met the entry criteria and formed the study sample. Patients with a Geriatric Depression Scale score of 7 or above were classified as having PSD. The presence and location of CMBs were evaluated with magnetic resonance imaging.
Results: Compared to the non-PSD group, patients with PSD were more likely to have pontine CMBs (32.0% vs 18.2%; P = .019). The presence of pontine CMBs remained an independent predictor of PSD in the multivariate analysis, with an odds ratio of 2.2 (P = .016).
Conclusion: The results suggest that pontine CMBs are associated with a higher risk of developing PSD.
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