Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, February 4, 2015

Consumption of fruit and vegetable and risk of coronary heart disease: a meta-analysis of prospective cohort studies

How much more information is needed before your hospital creates a post-stroke diet while in the hospital and for you to take home? I bet it will be 50 years.
My diet here: I'll have to add walnuts.


What would a post-stroke diet look like?


http://www.internationaljournalofcardiology.com/article/S0167-5273%2815%2900104-7/abstract?rss=yes
Publication stage: In Press Accepted Manuscript

Abstract

Background

Observational studies suggest that an association between fruit and vegetable consumption and coronary heart disease (CHD). However, the results are inconsistent. We conducted a meta-analysis to evaluate the relationship of fruit and vegetable consumption with CHD risk and quality the dose-response relationship between them.

Methods

Relevant prospective studies were identified by a search of PubMed, Embase and Web of Science databases to July 2014. A random-effects model was used to calculate the pooled relative risk (RR) and 95% confidence intervals (CI).

Results

Twenty-three studies involving 937,665 participants and 18,047 patients with CHD were included. Compared with the lowest consumption levels of total fruit and vegetable, fruit and vegetable, the RR of CHD was 0.84 (95%CI, 0.79–0.90), 0.86 (95%CI, 0.82–0.91), 0.87 (95%CI, 0.81–0.93), respectively. The dose-response analysis indicated that, the RR of CHD was 0.88 (95% CI: 0.85–0.91) per 477 g/day of total fruit and vegetable consumption, 0.84 (95% CI: 0.75–0.93) per 300 g/day of fruit intake and 0.82 (95% CI: 0.73–0.92) per 400 g/day of vegetable consumption. A nonlinear association of CHD risk with fruit or vegetable consumption separately was found (P for nonlinearity <.001). In the subgroup analysis of location, a significant inverse association was observed in Western populations, but not in Asian populations.

Conclusions

This meta-analysis indicates that total fruit and vegetable, fruit and vegetable consumption, are significantly associated with a lower risk of CHD. The significant inverse association was found in Western populations, but not in Asian populations, which warrants further research.

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