Interesting that they don't seem to be following the FDA guidelines on high dose statins. So ask your doctor how to reverse atherosclerosis naturally. I've written about a number of ways to do that. If your doctor doesn't know, you need to fire him/her and call the hospital president for continuing to employ doctors that aren't up-to-date on current practices. It's time to go to the mat on this, we have to remove all incompetent doctors.
http://www.ncbi.nlm.nih.gov/pubmed/25190674
Stegman B1,
Puri R2,
Cho L1,
Shao M3,
Ballantyne CM4,
Barter PJ5,
Chapman MJ6,
Erbel R7,
Libby P8,
Raichlen JS9,
Uno K1,
Kataoka Y10,
Nissen SE2,
Nicholls SJ11.
Abstract
OBJECTIVE:
Although
statins can induce coronary atheroma regression, this benefit has yet
to be demonstrated in diabetic individuals. We tested the hypothesis
that high-intensity statin therapy may promote coronary atheroma
regression in patients with diabetes.
RESEARCH DESIGN AND METHODS:
The
Study of Coronary Atheroma by Intravascular Ultrasound: Effect of
Rosuvastatin Versus Atorvastatin (SATURN) used serial intravascular
ultrasound measures of coronary atheroma volume in patients treated with
rosuvastatin 40 mg or atorvastatin 80 mg for 24 months. This analysis
compared changes in biochemistry and coronary percent atheroma volume
(PAV) in patients with (n = 159) and without (n = 880) diabetes.
RESULTS:
At
baseline, patients with diabetes had lower LDL cholesterol (LDL-C) and
HDL cholesterol (HDL-C) levels but higher triglyceride and CRP levels
compared with patients without diabetes. At follow-up, diabetic patients
had lower levels of LDL-C (61.0 ± 20.5 vs. 66.4 ± 22.9 mg/dL, P = 0.01)
and HDL-C (46.3 ± 10.6 vs. 49.9 ± 12.0 mg/dL, P < 0.001) but higher
levels of triglycerides (127.6 [98.8, 163.0] vs. 113.0 mg/dL [87.6,
151.9], P = 0.001) and CRP (1.4 [0.7, 3.3] vs. 1.0 [0.5, 2.1] mg/L, P =
0.001). Both patients with and without diabetes demonstrated regression
of coronary atheroma as measured by change in PAV (-0.83 ± 0.13 vs.
-1.15 ± 0.13%, P = 0.08). PAV regression was less in diabetic compared
with nondiabetic patients when on-treatment LDL-C levels were >70
mg/dL (-0.31 ± 0.23 vs. -1.01 ± 0.21%, P = 0.03) but similar when LDL-C
levels were ≤70 mg/dL (-1.09 ± 0.16 vs. -1.24 ± 0.16%, P = 0.50).
CONCLUSIONS:
High-intensity
statin therapy alters the progressive nature of diabetic coronary
atherosclerosis, yielding regression of disease in diabetic and
nondiabetic patients.
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