Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, June 23, 2016

The Role of Spasticity in Functional Neurorehabilitation- Part I: The Pathophysiology of Spasticity, the Relationship with the Neuroplasticity, Spinal Shock and Clinical Signs

While a great explanation of what spasticity is I could get nothing out of this to alleviate spasticity.
http://www.archivesofmedicine.com/medicine/the-role-of-spasticity-in-functional-neurorehabilitation-part-i-the-pathophysiology-of-spasticity-the-relationship-with-the-neurop.pdf

Angela Martins
*
Department of Veterinary Science, Lusophone University of Humanities and Technology, Hospital Veterinário da Arrábida, Portugal
*
Corresponding author:
Angela
Martins,
Department of Veterinary Science, Lusophone University of Humanities and Technology, Hospital
Veterinário da Arrábida, Portugal, Tel: 212181441; E-mail: vetarrabida.lda@gmail.com
Rec date:
Feb 29, 2016;
Acc date:
Apr 05, 2016;
Pub date:
April 12, 2016
Copyright:
© 2016
Martins
A. This is an open-access article distributed under the terms of the Creative Commons Attribution
License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
Citation:
Martins
A. The Role of Spasticity in Functional Neurorehabilitation-
Part I: The Pathophysiology of Spasticity, the Relationship with the Neuroplasticity,
Spinal Shock and Clinical Signs. Arch Med. 2016, 8:3
Abstract
The symptom/clinical sign of spasticity is extremely important in functional neurorehabilitation, since it reduces the functional independence both in the
quadruped animal as in the human biped.  This clinical sign/symptom manifests itself alongside with pain, muscle weakness, impaired coordination and poor motor planning, leading to a spastic movement disorder.
To perform a correct FNR protocol an understanding of its pathophysiology is required. In
addition FNR often stimulates the property of the central nervous system,
which is neuroplasticity, which may potentiate the spastic movement disorder.
In this regard, especially in the human biped, we must take into account the appearance of spinal shock and its development into spastic movement disorder, and therefore, a
tight and constant monitoring of clinical signs is essential in order to choose the adequate methods and modalities of FNR

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