Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Monday, April 3, 2017

Aerobic fitness is associated with low cardiovascular disease risk: The impact of lifestyle on early risk factors for atherosclerosis in young healthy Swedish individuals - The Lifestyle, Biomarker, and Atherosclerosis study

I was incredibly fit and still suffered an ischemic stroke, of course the carotid dissection probably occurred during exertions on my 6 day, 21 mile whitewater canoe trip, with portages up to 1.5 miles. My extreme fitness caused my stroke but also probably saved my life after the stroke occurred.
https://www.mdlinx.com/internal-medicine/medical-news-article/2017/03/30/cimt-cholesterol-insulin-resistance-body-fat/7108753/?
Vascular Health and Risk Management
Fernstrom M, et al.
The objectives of the study portrayed in this paper were to evaluate selected cardiometabolic biomarkers, carotid intima–media thickness (cIMT) as a marker of subclinical atherosclerosis, and lifestyle–associated indicators (food habits, handgrip strength, and oxygen uptake, VO2 max); examine the relationship between cIMT and lifestyle factors; and recognize subjects at risk of cardiovascular disease (CVD) utilizing a risk score and to compare the characteristics of subjects with and without risk of CVD. High aerobic fitness is related to low CVD risk in Swedish young adults. The high prevalence of young adults observed with unfavorable levels of high–density lipoprotein cholesterol and homeostasis model assessment of insulin resistance raises concerns about future CVD risk.

Methods

  • They collected blood samples in a fasting state, and food habits were reported through a questionnaire.
  • In this study, cIMT was measured by ultrasound, and VO2 max was measured by ergometer bike test.
  • The risk score was calculated according to Wildman.

Results

  • In this study, they found cIMT (mean ± standard deviation) was 0.50±0.06 mm, and VO2 max values were 37.8±8.5 and 42.9±9.9 mL/kg/min, in women and men, respectively.
  • No correlation was found between aerobic fitness expressed as VO2 max (mL/kg/min) and cIMT.
  • By using Wildman’s definition, 12% of the subjects were classified as being at risk of CVD, and 15% had homeostasis model assessment of insulin resistance.
  • An aggregate of 35% of women and 25% of men had lower high–density lipoprotein cholesterol than suggested.
  • Food habits did not differ between those at risk and those not at risk.
  • However, aerobic fitness measured as VO2 max (mL/kg/min) differed; 47% of the subjects at risk had low aerobic fitness compared to 23% of the nonrisk subjects (P<0.001).

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