Deans' stroke musings

Changing stroke rehab and research worldwide now.Time is Brain!Just think of all the trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 493 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It's quite disgusting that this information is not available from every stroke association and doctors group.
My back ground story is here:http://oc1dean.blogspot.com/2010/11/my-background-story_8.html

Saturday, April 29, 2017

Does atrial fibrillation cause cognitive decline and dementia?

You'll have to have your doctor fix your AF and prevent your cognitive decline and dementia.
Or is the problem that fixing AF causes decline and dementia? Cause and effect analysis needed.
https://www.mdlinx.com/internal-medicine/medical-news-article/2017/04/26/atrial-fibrillation-dementia-alzheimer-s-disease/7127317/?

EP Europace
Dietzel J, et al.
Researchers assessed the impact of atrial fibrillation (AF) on cognitive function. They gathered and summarized the published literature data on the association between AF and cognitive decline and found evidence supporting the notion that AF independently confers risk for cognitive decline and dementia in general and for Alzheimer’s disease in particular. The possible mechanisms involved were: AF–related ischaemic stroke, white matter damage and systemic inflammation.

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