Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, February 1, 2018

Tissue Plasminogen Activator Provides No Benefit for Mild Strokes

Oh God, another too good to treat review. Your solution is to do nothing or aspirin? No smaller bolus delivered by magnetic direction? 
http://dgnews.docguide.com/tissue-plasminogen-activator-provides-no-benefit-mild-strokes?
January 30, 2018
By Alex Morrisson
LOS ANGELES -- January 30, 2018 -- Treatment of mild strokes with tissue plasminogen activator (tPA) may cause more harm than good, according to a study presented here at the 2018 International Stroke Conference (ISC).
In the PRISM trial, 78.2% of patients who experienced a stroke but did not have clearly disabling deficits and were treated with alteplase were discharged with modified Rankin Scores of 0-1. However, if these patients were treated with aspirin, 81.5% were discharged with modified Rankin Scores of 0-1.
Symptomatic intracranial haemorrhage in the trial was experienced by 5 of the 154 (3.2%) patients on alteplase compared with zero symptomatic intracranial haemorrhages in the 153 patients who were assigned to aspirin.
“Among patients with low National Institutes of Health Stroke Scale scores and not clearly-disabling deficits, alteplase may not provide benefit and increases the risk of intracranial haemorrhage,” said Pooja Khatri, MD, University of Cincinnati, Cincinnati, Ohio.
Patients in the study were randomised to receive alteplase 0.9 mg/kg plus aspirin 325 mg or aspirin alone. The intravenous drug had to be administered within 3 hours of the last time the person was known well. Patients were ineligible for the study if they had a modified Rankin Score of 2-5 prior to having the stroke. The primary endpoint was functional outcome (Rankin score of 0-1 at 90 days).
The PRISM trial was truncated due to slow enrolment. The researchers enrolled 313 patients, shy of the 948 patients that were considered necessary to determine the role of alteplase in the the treatment algorithm for stroke. The researchers analysed in an intent-to-treat method the 156 patients in the alteplase group and 157 in the aspirin only group.
“The benefits of alteplase are not likely to extend to those without clearly-disabling deficits at presentation,” said Dr. Khatri. “These findings should not be extrapolated to patients with NIHSS 0-5 scores and clearly-disabling strokes.”
[Presentation title: Alteplase for the Treatment of Acute Ischemic Stroke in Patients With Low NIHSS and Not Clearly-Disabling Deficits: Primary Results of the PRISMS Trial. Abstract LB9]

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