Deans' stroke musings

Changing stroke rehab and research worldwide now.Time is Brain!Just think of all the trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 493 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It's quite disgusting that this information is not available from every stroke association and doctors group.
My back ground story is here:http://oc1dean.blogspot.com/2010/11/my-background-story_8.html

Wednesday, April 25, 2018

Study finds continued birth of new neurons (neurogenesis) well into our 70s

Does this refute this research from March 2018? What is your doctors' protocol to create neurogenesis? Or are your doctors sitting on their asses waiting for SOMEONE ELSE TO SOLVE THE PROBLEM?

Human hippocampal neurogenesis drops sharply in children to undetectable levels in adults March 2018

What protocol is your doctor have you doing to create neurogenesis? PROTOCOL,  not a guideline. If no protocol is given you have incompetency in action.

Study finds continued birth of new neurons (neurogenesis) well into our 70s

Well into our 70s, we continue to develop new cells in an area of the brain responsible for new memories and exploration of new environments, scientists report.
These new brain cells sustain our abilities to make new memories, learn, and cope with the environment, and they are important for emotional responses,” Dr. Maura Boldrini from Columbia University in New York City told Reuters Health by email…Even the oldest brains produced new brain cells. The number of developing and immature brain cells remained stable across the age range, the researchers reported in the journal Cell Stem Cell.
There was, however, a decline in the ability of mature nerve cells to change their function — a property known as neuroplasticity — with increasing age… “We know that vasculature can become weaker with aging, and we need to find ways to keep our (blood vessels) healthy so that our brain can remain more plastic,” Dr. Boldrini said. “This means that through healthy lifestyle, enriched environment, social interactions, and exercise” — all of which help maintain healthy blood vessels — “we can maintain these neurons healthy and functioning and sustain healthy aging.”

The Study:

  • Summary: Adult hippocampal neurogenesis declines in aging rodents and primates. Aging humans are thought to exhibit waning neurogenesis and exercise-induced angiogenesis, with a resulting volumetric decrease in the neurogenic hippocampal dentate gyrus (DG) region, although concurrent changes in these parameters are not well studied. Here we assessed whole autopsy hippocampi from healthy human individuals ranging from 14 to 79 years of age. We found similar numbers of intermediate neural progenitors and thousands of immature neurons in the DG, comparable numbers of glia and mature granule neurons, and equivalent DG volume across ages. Nevertheless, older individuals have less angiogenesis and neuroplasticity and a smaller quiescent progenitor pool in anterior-mid DG, with no changes in posterior DG. Thus, healthy older subjects without cognitive impairment, neuropsychiatric disease, or treatment display preserved neurogenesis. It is possible that ongoing hippocampal neurogenesis sustains human-specific cognitive function throughout life and that declines may be linked to compromised cognitive-emotional resilience.

The Study in Context:

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