Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, June 2, 2016

Neurogenesis in the Developing and Adult Brain—Similarities and Key Differences

How is your doctor going to use this to update your neurogenesis protocol? WHEN?
If not in the next couple of weeks you need to fire that doctor. We need to weed the incompetent doctors out of treating patients.
http://cshperspectives.cshlp.org/content/early/2016/05/27/cshperspect.a018853.abstract 
 
  1. David Petrik1,2
+ Author Affiliations
  1. 1Institute of Stem Cell Research, Helmholtz Center Munich, 85764 Neuherberg, Munich, Germany
  2. 2Physiological Genomics, Biomedical Center, Ludwig-Maximilians-University, 80336 Munich, Germany
  3. 3Synergy, Munich Cluster for Systems Neurology, 81377 Munich, Germany
  4. 4Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45140
  5. 5Departments of Pediatrics and Neurosurgery, University of Cincinnati College of Medicine, Cincinnati, Ohio 45267
  1. Correspondence: magdalena.goetz@helmholtz-muenchen.de; masato.nakafuku@cchmc.org

Abstract

Adult neurogenesis in the mammalian brain is often viewed as a continuation of neurogenesis at earlier, developmental stages. Here, we will critically review the extent to which this is the case highlighting similarities as well as key differences. Although many transcriptional regulators are shared in neurogenesis at embryonic and adult stages, recent findings on the molecular mechanisms by which these neuronal fate determinants control fate acquisition and maintenance have revealed profound differences between development and adulthood. Importantly, adult neurogenesis occurs in a gliogenic environment, hence requiring adult-specific additional and unique mechanisms of neuronal fate specification and maintenance. Thus, a better understanding of the molecular logic for continuous adult neurogenesis provides important clues to develop strategies to manipulate endogenous stem cells for the purpose of repair.

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