Deans' stroke musings

Changing stroke rehab and research worldwide now.Time is Brain!Just think of all the trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 493 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It's quite disgusting that this information is not available from every stroke association and doctors group.
My back ground story is here:http://oc1dean.blogspot.com/2010/11/my-background-story_8.html

Thursday, January 12, 2017

Link Found Between Concussions, Alzheimer's

Be careful out there. What is your doctor doing to increase your cortical thickness?
http://www.rdmag.com/news/2017/01/link-found-between-concussions-alzheimers?

A direct link has been found between concussions and Alzheimer’s disease.
New research by the Boston University School of Medicine (BUSM) found that concussions will accelerate brain atrophy and cognitive decline in people who are already at a genetic risk for the disease.
The researchers studied 160 Iraq and Afghanistan war veterans, some of which had suffered one or more concussions and some who were concussion-free. By using MRI imaging they measured the thickness of the cerebral cortex in seven regions that are the first to show atrophy in Alzheimer’s sufferers, as well as seven control regions.
Jasmeet Hayes, Ph.D., assistant professor of psychiatry at BUSM and research psychologist at the National Center for PTSD, VA Boston Healthcare System, explained the results of the study.
“We found that having a concussion was associated with lower cortical thickness in brain regions that are the first to be affected in Alzheimer's disease,” Hayes said in a statement. “Our results suggest that when combined with genetic factors, concussions may be associated with accelerated cortical thickness and memory decline in Alzheimer's disease relevant areas.”
While it is unclear whether mild traumatic brain injuries or concussions increase the risk for developing neurodegenerative diseases, moderate-to-severe traumatic brain injuries have been proven to be one of the strongest environmental risk factors.
One of the most noteworthy results of the study is brain abnormalities were found in a younger group than what was expected, with the average age being 32 years old.
“These findings show promise for detecting the influence of concussion on neurodegeneration early in one's lifetime, thus it is important to document the occurrence and subsequent symptoms of a concussion, even if the person reports only having their ‘bell rung’ and is able to shake it off fairly quickly, given that when combined with factors such as genetics, the concussion may produce negative long-term health consequences," Hayes said.
With these results, the researchers will work to find the precise concussion-related mechanism that accelerate the onset of Alzheimer’s disease, chronic traumatic encephalopathy, Parkinson’s and other neurodegenerative diseases.
According to Hayes, treatments may one day be developed to target those mechanisms and delay the onset of neurodegenerative pathology.

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