I know cause and effect haven't been totally proven yet but I know exactly what I'm telling my doctor to do. I'm not dying from this, I survived a pretty large stroke, this is not going to kill me.
COVID ‐19: a global threat to the nervous system
First published: 07 June 2020
This article has been accepted for publication and
undergone full peer review but has not been through the copyediting,
typesetting, pagination and proofreading process, which may lead to
differences between this version and the Version of Record. Please cite
this article as doi: 10.1002/ana.25807.
Abstract
In less than 6 months, the severe acute respiratory
syndrome‐coronavirus type 2 (SARS‐CoV‐2) has spread worldwide infecting
nearly 6 million people and killing over 350,000. Initially thought to
be restricted to the respiratory system, we now understand that
coronavirus disease 2019 (COVID‐19) also involves multiple other organs
including the central and peripheral nervous system. The number of
recognized neurologic manifestations of SARS‐CoV‐2 infection is rapidly
accumulating. These may result from a variety of mechanisms including
virus‐induced hyper‐inflammatory and hypercoagulable states, direct
virus infection of the CNS, and post‐infectious immune mediated
processes. Example of COVID‐19 CNS disease include encephalopathy,
encephalitis, acute disseminated encephalomyelitis, meningitis, ischemic
and hemorrhagic stroke, venous sinus thrombosis and endothelialitis. In
the peripheral nervous system COVID‐19 is associated with dysfunction
of smell and taste, muscle injury, the Guillain‐Barre syndrome and its
variants. Due to its worldwide distribution and multifactorial
pathogenic mechanisms, COVID‐19 poses a global threat to the entire
nervous system. While our understanding of SARS‐CoV‐2 neuropathogenesis
is still incomplete and our knowledge is evolving rapidly, we hope that
this review will provide a useful framework and help neurologists in
understanding the many neurologic facets of COVID‐19.
No comments:
Post a Comment