Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, August 30, 2025

Impact of Short‐Term Exposure to Ozone on Hospital Admissions for Multiple Cardiovascular Diseases: A Systematic Review and Meta‐Analysis

My God, even the AHA doesn't know that stroke has been called neurological disease by the WHO since 2006 not cardiovascular, once again proving the stroke medical world doesn't keep up to date in their field!

The latest here:

Impact of Short‐Term Exposure to Ozone on Hospital Admissions for Multiple Cardiovascular Diseases: A Systematic Review and Meta‐Analysis


Yu You MM,  Juan Chen PhD,  Kai Wang MM, Tong Wu MM, Lijun Bai MM, Ge Li MM https://orcid.org/0000-0001-6659-0291, and Shaowei Wu PhD https://orcid.org/0000-0003-2680-6064 shaowei_wu@xjtu.edu.cnAuthor Info & Affiliations Journal of the American Heart Association New online https://doi.org/10.1161/JAHA.124.037205>

Abstract

Background
The associations between short‐term exposure to ambient ozone (O3) pollution and the risks of hospital admissions (an indicator for morbidity) for different subtypes of cardiovascular disease (CVD) remain unclear. Therefore, this study aimed to evaluate the associations between short‐term exposure to O3 and the risks of hospital admissions for all‐cause CVD, arrhythmia, atrial fibrillation, cardiac diseases, heart failure, ischemic heart disease, myocardial infarction (MI), and stroke.
Methods
Four electronic databases, including PubMed, Web of Science, Scopus, and Embase, were searched for articles published up to December 21, 2023. Epidemiological studies reporting the associations between short‐term exposure to O3 and the risks of hospital admissions for CVDs were included on the basis of stringent inclusion and exclusion criteria. The random‐effect model was applied to estimate pooled relative risk and 95% CIs. To identify potential effect modifiers, subgroup and meta‐regression analyses were also performed. A total of 111 articles with >65 million participants were included in the meta‐analysis. For a 10 μg/m3 increase in short‐term exposure to O3, the relative risks of hospital admissions for MI and stroke were 1.0084 (95% CI, 1.0019–1.0151) and 1.0023 [95% confidence interval (CI), 1.0012–1.0034], respectively. Subgroup and meta‐regression analyses revealed that study area and national economic status could influence the impact of O3 on different subtypes of CVD.This meta‐analysis provides compelling evidence for the adverse effects of short‐term exposure to ambient O3 pollution on the risks of hospital admissions for MI and stroke
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