Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, May 7, 2013

Simultaneous Characterizations of Reflex and nonreflex, dynamic and static changes in spastic hemiparesis

I'm sure your doctor can decipher this and apply it to your stroke protocol.
http://jn.physiology.org/content/early/2013/04/27/jn.00573.2012.abstract

Abstract

This study characterizes tonic and phasic stretch-reflex and stiffness and viscosity changes associated with spastic hemiparesis. Perturbations were applied to the ankle of 27 hemiparetic and 36 healthy subjects under relaxed or active contracting conditions. A nonlinear delay differential equation model characterized phasic and tonic stretch-reflex gains, elastic stiffness, and viscous damping. Tendon reflex was characterized with reflex gain and threshold. Reflexively, tonic-reflex gain was increased in spastic ankles at rest (P&lt0.038) and was not regulated with muscle contraction, indicating impaired tonic stretch-reflex. Phasic-reflex gain in spastic plantarflexors was higher and increase faster with plantarflexor contraction (P&lt0.012) than controls (P&lt0.023) and higher in dorsi-flexors at lower torques (P&lt0.038), primarily due to its increase at rest (P=0.045), indicating exaggerated phasic stretch-reflex especially in more spastic plantarflexors which showed higher phasic stretch-reflex gain than dorsi-flexors (P&lt0.032). Spasticity was associated with increased tendon-reflex gain (P=0.002) and decreased threshold (P&lt0.001). Mechanically, stiffness in spastic ankles was higher than that in controls across plantar/dorsi-flexion torque levels (P&lt0.032), and the more spastic plantarflexors were stiffer than dorsi-flexors at comparable torques (P&lt0.031). Increased stiffness in spastic ankles was mainly due to passive stiffness increase (P&lt0.001), indicating increased connective tissues/shortened fascicles. Viscous damping in spastic ankles was increased across the plantar flexion torque levels and at lower dorsi-flexion torques, reflecting increased passive viscous damping (P=0.033). The more spastic plantar flexors showed higher viscous damping than dorsi-flexors at comparable torque levels (P&lt0.047). Simultaneous characterizations of reflex and nonreflex changes in spastic hemiparesis may help evaluate/treat them more effectively.

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1 comment:

  1. Rebecca DuttonMay 8, 2013 at 9:42 AM

    You don't need high-tect equipment to feel these responses to movement. Therapists have known all this for decades. What a waste of research resources.

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