Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, July 27, 2016

What can be done when mild cognitive impairment occurs?

I got nothing useful out of this, so you will have to ask your personal stroke researcher how to use this information. It should be done once by a great stroke association but instead we have fucking failures of stroke associations. And will need to be done for 10 million yearly stroke survivors.

What can be done when mild cognitive impairment occurs? 


A randomized controlled study has evaluated the effects of two treatments for mild cognitive impairment. Authors examined the efficacy of group-based cognitive intervention (GCI) and home-based cognitive intervention (HCI) in amnestic mild cognitive impairment (aMCI) and intervention effects on serum brain-derived neurotrophic factor (BDNF). Methods: In this randomized and rater-blinded trial, 293 patients with aMCI from 18 nationwide hospitals were randomized as follows: 96 to the GCI group, 98 to the HCI group and 99 to the control group.
For 12 weeks, participants receiving GCI participated twice per week in group sessions led by trained instructors, and those receiving HCI completed homework materials 5 days per week. They were assessed at baseline, post intervention (PI) and at the 6-month follow-up. The primary endpoint was the change from baseline to PI in the modified Alzheimer's Disease Assessment Scale-cognitive subscale (ADAS-Cog). Results showed that compared to controls (a 0.8-point decrease), subjects receiving GCI (a 2.3-point decrease, p = 0.01) or HCI (a 2.5-point decrease, p = 0.02) reported significant improvements in the modified ADAS-Cog at PI, respectively. These findings were confirmed at 6-month follow-up, where those receiving GCI or HCI still had better scores in the modified ADAS-Cog than controls.
In addition, changes in BDNF levels significantly correlated with the changes in the modified ADAS-Cog in the GCI (r = -0.29, p = 0.02 at PI) and HCI (r = -0.27, p = 0.03 at 6-month follow-up) groups. Authors concluded that an enhanced brain plasticity may be a crucial component of the mechanism underpinning cognitive improvements associated with cognitive interventions.

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