Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, August 24, 2017

Non-Invasive Eye Scan Could Detect Signs of Alzheimer’s Years Before Patients Show Symptoms

After your doctor runs this test, what are the EXACT effective dementia prevention protocols you are prescribed?

1. A documented 33% dementia chance post-stroke from an Australian study?   May 2012.
2. Then this study came out and seems to have a range from 17-66%. December 2013.
3. A 20% chance in this research.   July 2013.

Non-Invasive Eye Scan Could Detect Signs of Alzheimer’s Years Before Patients Show Symptoms 

August 18, 2017
LOS ANGELES -- August 18, 2017 -- Alzheimer’s disease affects the retina similarly to the way it affects the brain, according to a study published in JCI Insight.
The study also revealed that an investigational, non-invasive eye scan could detect the key signs of Alzheimer’s disease years before patients experience symptoms.
Using a high-definition eye scan developed especially for the study, researchers detected the crucial warning signs of Alzheimer’s disease: amyloid-beta deposits. The findings represent a major advancement toward identifying people at high risk for the debilitating condition years sooner.
“The findings suggest that the retina may serve as a reliable source for Alzheimer’s disease diagnosis,” said Maya Koronyo-Hamaoui, PhD, Cedars-Sinai Medical Center, Los Angeles, California. “One of the major advantages of analysing the retina is the repeatability, which allows us to monitor patients and potentially the progression of their disease.”
Another key finding from the study was the discovery of amyloid plaques in previously overlooked peripheral regions of the retina. The researchers found that the amount of plaque in the retina correlated with plaque amount in specific areas of the brain.
“Now we know exactly where to look to find the signs of Alzheimer’s disease as early as possible,” said Yosef Koronyo, Cedars-Sinai Medical Center.
“Our hope is that eventually the investigational eye scan will be used as a screening device to detect the disease early enough to intervene and change the course of the disorder with medications and lifestyle changes,” said Keith L. Black, MD, Cedars-Sinai Medical Center.
The study included 16 patients with Alzheimer’s disease. The patients drank a solution that included curcumin, which causes amyloid plaque in the retina to light up and be detected by a modified scanning laser ophthalmoscope. The patients were then compared to a group of 37 younger, cognitively normal individuals.
Histological examination uncovered classical and neuritic-like Abeta deposits with increased retinal amyloid-beta 42 plaques (4.7-fold; P = .0063) and neuronal loss (P = .0023) in patients with AD versus matched controls. Retinal amyloid-beta plaque mirrored brain pathology, especially in the primary visual cortex.
Reference: https://doi.org/10.1172/jci.insight.93621
SOURCE: Cedars-Sinai Medical Center

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