Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, October 2, 2018

Antioxidant Reduces Risk for 2nd Stroke, Heart Attack

Now YOU just need to find the protocol for this and train your doctor in its use.  Not the other way around. Has your doctor EVER  put ANY clinical research into practice? This is one of the few things I got from the NSA that was worth it.

Antioxidant Reduces Risk for 2nd Stroke, Heart Attack


Posted by Lynn Bronikowski Sep 27 2018
Healthcare professionals have long known that in the months after a heart attack or stroke, patients are more likely to have another attack or stroke.
Now a study published in the Journal of the American College of Cardiology explains what happens inside blood vessels to increase stroke and heart attack risk—and suggests a new way to treat it.
Researchers found that heart attacks in mice caused inflammatory cells and platelets to more easily stick to the inner lining of arteries throughout the body—particularly where there was already plaque, according to the study’s writeup. As a result, these sticky cells and platelets caused plaque to become unstable and contribute to blood clots that led to another stroke or heart attack.
But the study found treating mice that had experienced a stroke or heart attack with the powerful antioxidant apocynin cut plaque buildup in half and lowered inflammation to pre-attack levels.
"Knowing that newer forms of antioxidants such as apocynin can lower the risk of a second heart attack or stroke gives us a new treatment to explore and could one day help reduce heart attacks and strokes," said the study’s author, Jonathan R. Lindner, M.D., a professor of cardiovascular medicine at the OHSU School of Medicine.
Lindner penned the research paper with colleagues from OHSU, Scripps Research Institute and Bloodworks NW.
The researchers discovered the sticky cells and platelets by using unique forms of ultrasound imaging they developed to view molecules on the lining of blood vessels.
This research could help explain why the recent Canakinumab Anti-inflammatory Thrombosis Outcomes Study, also known as the CANTOS clinical trial, found an anti-inflammatory drug already approved to treat juvenile arthritis also reduced the risk of a second heart attack in trial participants by 15 percent.
Lindner and his colleagues are further studying how the relative stickiness of remote arteries affects the risks for additional heart attacks and strokes and are also evaluating new therapies beyond antioxidants.
The study was supported by the National Institutes of Health and the Swiss National Science Foundation.

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