Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Friday, June 21, 2019

Associations of amyloid, tau, and neurodegeneration biomarker profiles with rates of memory decline among individuals without dementia

You need to have your doctor test for these biomarkers because of your likely chance of dementia. Then you need your doctor's SPECIFIC DEMENTIA PREVENTION PROTOCOLS applied.  No protocol, fire that doctor.

1. A documented 33% dementia chance post-stroke from an Australian study?   May 2012.

2. Then this study came out and seems to have a range from 17-66%. December 2013.

3. A 20% chance in this research.   July 2013.

4. Dementia Risk Doubled in Patients Following Stroke September 2018

 

Associations of amyloid, tau, and neurodegeneration biomarker profiles with rates of memory decline among individuals without dementia

JAMAJack CR, et al. | June 20, 2019

Via performing a longitudinal cohort study that included 480 participants without dementia (aged 60 years or older), researchers ascertained the associations between amyloid, tau, and neurodegeneration biomarker profiles and memory decline, and determined if biomarkers rendered incremental prognostic value beyond more readily available clinical and genetic information. Among older persons without baseline dementia, when followed up for a median of 4.8 years, a little but statistically meaningful improvement in the prediction of memory decline over a model with more readily available clinical and genetic variables was observed through a prediction model that included amyloid positron emission tomography (PET), tau PET, and magnetic resonance imaging cortical thickness. However, the uncertainty of the clinical importance of this difference was noted.
Read the full article on JAMA

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