Useless, NOTHING ON STROKE PROTOCOLS FOR USE. And even worse you ask for followup to cover up your failures in your research.
Maybe these might help you talk to your doctor.
- 1Department of Neurology, Xuanwu Hospital, Capital Medical University, Beijing, China
- 2Department of Neurosurgery, School of Medicine, Wayne State University, Detroit, MI, United States
- 3Department of Neurosurgery, Xuanwu Hospital, Capital Medical University, Beijing, China
- 4Center of Stroke, Beijing Institute for Brain Disorders, Beijing, China
As we all know that stroke is still a leading cause of death and
acquired disability. Etiological treatment and brain protection are
equally important. This review aimed to summarize the advance of
normobaric-hyperoxia (NBHO) on brain protection in the setting of
experimental stroke and brain trauma. We analyzed the data from relevant
studies published on PubMed Central (PMC) and EMBASE, about NBHO on
brain protection in the setting of experimental ischemic and hemorrhagic
strokes and brain trauma, which revealed that NBHO had important value
on improving hypoxia and attenuating ischemia damage. The mechanisms of
NBHO involved increasing the content of oxygen in brain tissues,
restoring the function of mitochondria, enhancing the metabolism of
neurons, alleviating blood-brain barrier (BBB) damage, weakening brain
cell edema, reducing intracranial pressure, and improving cerebral blood
flow, especially in the surrounding of injured area of the brain, to
make the neurons in penumbral area alive. Compared to hyperbaric oxygen
(HBO), NBHO is more safe and more easily to transform to clinical use,
whereby,
further studies about the safety and efficacy as well as the
proper treatment protocol of NBHO on human
may be still needed.
Zhiying Chen
Yuchuan Ding
Xunming Ji
Ran Meng
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