Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Monday, November 24, 2025

Supplement Shows Promise in Blocking Amyloid in Alzheimer’s

Your incompetent? doctor and hospital won't get human testing going, will they?

All this earlier research which I bet your doctor/hospital knows nothing!

  • arginine (2 posts to December 2016)
  • arginine-rich peptides (1 post to February 2015)
  • Cationic arginine-rich peptides (1 post to March 2020)
  • poly-arginine peptides (1 post to February 2015)
  • Do you prefer your doctor and hospital incompetence NOT KNOWING? OR NOT DOING?

    Supplement Shows Promise in Blocking Amyloid in Alzheimer’s

    Summary: A new study shows that oral arginine, a naturally occurring amino acid, can significantly suppress amyloid-β aggregation in Alzheimer’s disease models. Researchers found that arginine not only prevented Aβ42 from clumping but also reduced plaques and inflammation in mouse and fruit fly models carrying Alzheimer’s-related mutations.

    Treated mice showed improved cognitive performance alongside reduced neuroinflammation, indicating broad neuroprotective effects. Because arginine is clinically safe, affordable, and widely available, it holds strong potential as a repurposed therapeutic strategy pending further human studies.

    Key Facts:

    • Aggregation Blocker: Arginine reduced Aβ42 aggregation in vitro and in Alzheimer’s disease models.
    • Neuroprotective Effects: Treatment lowered plaques, decreased inflammatory markers, and improved behavior.
    • Clinical Potential: Arginine’s safety and low cost make it a promising candidate for rapid therapeutic repurposing.

    Source: Kindai University

    Alzheimer’s disease (AD), a progressive neurodegenerative disorder, is one of the leading causes of dementia worldwide, and currently has no definitive cure. Although antibody-based therapies that target amyloid β (Aβ) have recently been developed, their clinical effectiveness remains limited. 

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