Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, November 12, 2013

PET Imaging Technique Finds Plaques 'Ripe for Rupture'

This is probably what I should have had done after my dad was diagnosed with 85% blockage in his left carotid artery. But that would have required his doctor being trained well enough to know that blockage might be hereditary.

PET Imaging Technique Finds Plaques 'Ripe for Rupture'


Positron-emission tomography (PET) can identify recently ruptured atherosclerotic plaques or those at risk of rupture in patients using the metabolic tracer 18F-sodium fluoride (18F-NaF), suggests a small prospective observational study[1].
Focal uptake of the tracer in the coronaries "informs about calcification activity, which is believed to be a healing response to intense plaque inflammation," according to the study's lead author, Dr Nikhil V Joshi (University of Edinburgh, Scotland). It was seen in the culprit artery of >90% of the group's patients with recent acute MI and in more than a third of those with stable coronary disease, he explained to heartwire .
Plaques with concentrations of increased tracer uptake showed features linked to rupture risk (positive remodeling, microcalcification, large necrotic core) at coronary computed tomography (CT) and intravascular ultrasound (IVUS), those findings supported by carotid endarterectomy tissue histology.

More at link and names for your doctor to talk to if needed.

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