Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, September 4, 2025

Excessive glutamate stimulation and neurodegeneration

 Your incompetent? doctor has done nothing on glutamate poisoning!
  • glutamate poisoning (7 posts to July 2020)
  • And I'm sure they DID NOTHING with this either!

    generic drug candesartan (brand name: ATACAND®) Blood Pressure Drug Helps Alzheimer's June 2018

     This line from there is instructive:

    The scientists found that candesartan prevented glutamate-induced neuronal death. 

    Absolute proof the entire stroke medical world is blitheringly stupid!

    Excessive glutamate stimulation and neurodegeneration


    Lena Kabulashvili, Mariam Pipia

    Abstract


    Glutamate is the main excitatory neurotransmitter in the central nervous system and plays a key role in synaptic transmission, learning, and memory processes. However, excessive glutamate release and overstimulation of excitatory receptors (NMDA, AMPA) lead to an overload of calcium ions in neurons. This, in turn, promotes the formation of free radicals, mitochondrial damage, and the activation of cellular death pathways.

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