Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, September 24, 2025

Boosting astrocytic NAD+ against tauopathy

 

Did your incompetent? doctor DO NOTHING with this earlier research?
  • NAD (3 posts to May 2014)

    • Has your incompetent board of directors not fired your doctor yet? Is 10 years of incompetence not enough to prove incompetence?

    Tauopathy is a group of neurodegenerative diseases characterized by the abnormal accumulation and aggregation of tau protein in the brain.

    Boosting astrocytic NAD+ against tauopathy

    Nature Aging (2025)Cite this article

    Nicotinamide adenine dinucleotide (NAD+) decline is a molecular characteristic of aging and age-associated neurodegenerative diseases. Lee and colleagues uncover an astrocyte-specific regulatory axis directed by the circadian clock component REV-ERBα, and provide proof-of-concept evidence that targeting this pathway alleviates tauopathy in mouse models.

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