Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Friday, November 16, 2012

Special issue commentary: The changing face of inflammation in the brain

For your doctor to know about, of course it probably needed to be known in the first days of your stroke but better luck next time in choosing your doctors.
http://www.sciencedirect.com/science/article/pii/S1044743112002047
Another phenomenon that has received very little attention is the matter of post stroke AC CE
PT ... model, but there is no doubt that we need more models in which neurons die in a ... Mechanisms
for transcellular diapedesis: probing and pathfinding by 'invadosome-like protrusions'.


Abstract

The study of Inflammation in the brain is a broad church and there is plenty of argument over semantics and the precise definition of what constitutes inflammation in such a diverse collection of pathologies. In this special issue, we sought to highlight the diversity of what is considered to be inflammation in the brain, and we have accepted that the presence of microglia cells with altered morphology remains a useful starting point. However, it is clear that whatever is the molecular expression profile that accompanies an activated microglia cell, it is not static and it is influenced by factors both intrinsic and extrinsic to the brain.

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