Use the labels in the right column to find what you want. Or you can go thru them one by one, there are only 27,840 posts. Searching is done in the search box in upper left corner. I blog on anything to do with stroke.DO NOT DO ANYTHING SUGGESTED HERE AS I AM NOT MEDICALLY TRAINED, YOUR DOCTOR IS, LISTEN TO THEM. BUT I BET THEY DON'T KNOW HOW TO GET YOU 100% RECOVERED. I DON'T EITHER, BUT HAVE PLENTY OF QUESTIONS FOR YOUR DOCTOR TO ANSWER.
Changing stroke rehab and research worldwide now.Time is Brain!trillions and trillions of neuronsthatDIEeach day because there areNOeffective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.
What this blog is for:
My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.
Saturday, January 18, 2020
Potential of coconut oil and medium chain triglycerides in the prevention and treatment of Alzheimer’s disease
Your doctor is responsible for elucidating the pros of coconut oil for this prevention/treatment. VS. this!
Brain glucose hypometabolism is a major early hallmark of AD.
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CO is a rich source of MCT, and ketone bodies arise from MCT metabolism.
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Ketone bodies serve as an alternative energy source for the glucose-deficient brain.
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CO, MCT and their derivatives show evidence of neuroprotective properties.
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CO, MCT and their derivatives show evidence of influencing AD related risk factors.
Abstract
Alzheimer’s
disease (AD) is the most common form of dementia. Currently, there is
no effective medication for the prevention or treatment of AD. This has
led to the search for alternative therapeutic strategies. Coconut
oil(CO) has a unique fatty acid composition that is rich in medium chain
fatty acids(MCFA), a major portion of which directly reaches the liver
via the portal vein, thereby bypassing the lymphatic system. Given that
brain glucose hypometabolism is a major early hallmark of AD, detectable
well before the onset of symptoms, ketone bodies from MCFA metabolism
can potentially serve as an alternative energy source to compensate for
lack of glucose utilisation in the brain. Additionally, neuroprotective
antioxidant properties of CO have been attributed to its polyphenolic
content. This review discusses how the metabolism of CO and MCFA may aid
in compensating the glucose hypometabolism observed in the AD brain.
Furthermore, we present the current evidence of the neuroprotective
properties of CO on cognition, amyloid-β pathogenicity, inflammation and
oxidative stress. The current review addresses the influence of CO/MCFA
on other chronic disorders that are risk factors for AD, and addresses
existing gaps in the literature regarding the use of CO/MCFA as a
potential treatment for AD.
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