Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Friday, September 30, 2016

The Drink That Really Is A Rapid Antidepressant

Never follow this until your doctor prescribes it for you. And that will be never.
http://www.spring.org.uk/2016/09/drink-really-rapid-antidepressant.php?omhide=true
The most well-known effect of this drink confirmed by research.
Alcohol produces the neural and molecular changes of a rapid antidepressant, new research concludes.
The drug lifts depression for up to 24 hours due to its effect on the brain’s chemistry.
Naturally, the study’s authors were quick to warn against self-medication.
Dr Kimberly Raab-Graham, the study’s first author, said:
“Because of the high comorbidity between major depressive disorder and alcoholism there is the widely recognized self-medication hypothesis, suggesting that depressed individuals may turn to drinking as a means to treat their depression.
We now have biochemical and behavioral data to support that hypothesis.”
The research was carried out in animals, but the effects seen were similar to other rapid antidepressants.
The behavioural effects were also similar to those seen in people.
 
Ketamine is another drug that has been recently shown to produce a rapid antidepressant effect.
Dr Raab-Graham said:
“There’s definitely a danger in self-medicating with alcohol.
There’s a very fine line between it being helpful and harmful, and at some point during repeated use self-medication turns into addiction.”
Doctors often advice against drinking alcohol while taking antidepressant medication.
It can cause a worsening of depression symptoms in the long-run.
Naturally, as this study demonstrates, people feel the benefit of drinking to their mood in the short-term.
Indeed, antidepressants can often increase the intoxicating effect of alcohol.
Repeatedly drinking too much, though, can clearly lead to a worse emotional state — especially the morning after.
Dr Raab-Graham said:
“Additional research is needed in this area, but our findings do provide a biological basis for the natural human instinct to self-medicate.
They also define a molecular mechanism that may be a critical contributor to the comorbidity that occurs with alcohol use disorder and major depressive disorder.”
The study was published in the journal Nature Communications (Wolfe et al., 2016).

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