Cerebral Damage after Stroke: The Role of Neuroplasticity as Key for Recovery

Oh fuck, dumping everything about recovery on the survivor with NO PROTOCOLS.  Useless.

The whole problem is that since your doctor did NOTHING to stop the 5 causes of the neuronal cascade of death in the first week. That would save billions of neurons. I only lost 5.4 billion neurons that first week because my doctor did nothing that first week. At $1000 a neuron that should have cost the hospital 5.4 trillion dollars. That would concentrate the hospital leadership on solving stroke. 

You are in a deep hole because of your doctor doing nothing the first week. Your doctor caused the problem, have them fix it. Screaming may be required.

Cerebral Damage after Stroke: The Role of Neuroplasticity as Key for Recovery

By Mubarak Muhammad and Tasneem Muhammad Hassan

Submitted: September 16th 2020Reviewed: December 16th 2020Published: January 19th 2021

DOI: 10.5772/intechopen.95512

Downloaded: 22

Abstract

Stroke remains global health care problem that constitutes world’s second-leading perpetrator of mortality and third most pronounced cause of all disabilities. The hallmark of cerebral stroke is the persistent loss of cerebral function consequence of abnormality of the blood supply. The ultimate goal of stroke care is to recover and maximize the cerebral functions lost due to the cerebral damage. Therefore, understanding the mechanism of cerebral damage after stroke is fundamental to comprehension of mechanisms of recovery following stroke, as well as key towards eliminating devastating human disability as a result of stroke. Therapeutic strategies aim to harness and enhance neuroplasticity offers reasonable level of hope towards maximizing recovery from post stroke impairments. This paper therefore, highlighted the mechanism of cerebral damage after stroke as well as elucidates the concept of neuroplasticity as key for recovery following stroke.