What this blog is for:
My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.
Amen, I have done research on this matter. and there are alternatives that can be used in nearly endless combinations. i have had written a few break through protocols on a similar but more emergent issue: global ischemic damage from a post anoxic event. The major etiology of my concern is cytotoxicity and oxidative damage that occurs during tissue reperfusion. In in a case of strokes of the transient nature the damage will likely culmilative dependind on the amount of time after re-oxygination and the amount of tissue that has been deprived of O2. Also the rate of reperfusion plays a role in rehabilition and eventual prognosis, that is if the attending has any idea of what is actually going on. You are on the right track with the insight of D-cycloserine which can be an adjunct to other drugs such as amantadine and other NMDA antagonists and modulators. All Cannot be place in a post due to not being published at this point, but look closer at glycine channel agonist and their role with other agents.Sorry no time to proof read the message should be rewritten later in light of this.
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