Wrong measurement, you measure the primary problem, inflammation, NOT this secondary issue. Some century from now, doctors will finally address the real problem, but you, your children and grandchildren will be dead.
Prevent inflammation and cholesterol won't be grabbed out of the bloodstream and packed into plaque. Solve the correct problem not a bystander. Don't listen to me, I'm not medically indoctrinated. Ask your doctor what the 25% of cholesterol in your body that is in your brain is doing. You can see by these dates that incompetence reigns supreme in the stroke medical world.
I'm sure there is much more good stuff in these posts:
inflammation (306 posts to December 2011)
I would much rather do this:
This gene could reduce inflammation after stroke June 2019
Or this:
Resolvins, generated naturally from fish oil, show promise as anti-inflammation treatment. Aug. 2016
Or this:
Antibody blocks inflammation, protects mice from hardened arteries and liver disease
June 2018
The latest here:
A comparison of two LDL cholesterol targets after ischemic stroke
New England Journal of Medicine — Amarenco P, Kim JS, Labreuche J, et al. | January 07, 2020
In this parallel-group trial carried
out in France and South Korea, researchers analyzed the target level
for LDL cholesterol to decrease cardiovascular events post-stroke. They
randomly assigned target LDL cholesterol levels below 70 mg per
deciliter (1.8 mmol per liter; lower-target group) or a target range of
90 mg to 110 mg per deciliter (2.3 to 2.8 mmol per liter; higher-target
group) in patients who suffered an ischemic stroke in the previous 3
months or a transient ischemic attack (TIA) within the previous 15 days.
In total, 2,860 patients were registered and followed for a median of
3.5 years; 1,430 were assigned to each LDL cholesterol target group.
There was no significant difference between the two groups in the
incidence of intracranial hemorrhage and newly diagnosed diabetes. The
authors discovered that patients with a target LDL cholesterol level of
< 70 mg per deciliter had a lower risk of subsequent cardiovascular
events after an ischemic stroke or TIA with evidence of atherosclerosis
than those with a target range of 90 mg to 110 mg per deciliter.
Read the full article on New England Journal of Medicine
No comments:
Post a Comment