Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, April 8, 2020

Functional Electrical Stimulation of Ankle Plantarflexor and Dorsiflexor Muscles: Effects on Poststroke Gait

In the 11 years since this came out, has a stroke protocol been published AND distributed  to ALL 10 million yearly stroke survivors? If not, THEN THERE IS COMPLETE FUCKING INCOMPETENCY IN TENS OF THOUSANDS OF STROKE DOCTORS AND THERAPISTS. Do you prefer your incompetency NOT KNOWING OR NOT DOING? Your Hobson's choice. Until we start writing and updating protocols stroke rehab will never move forward. Guidelines are useless because they leave leeway for the survivor to be blamed for not recovering. This is so obvious, IS EVERYONE IN STROKE LEADERSHIP COMPLETELY STUPID?

Functional Electrical Stimulation of Ankle Plantarflexor and Dorsiflexor Muscles: Effects on Poststroke Gait

 Stroke
. 2009 December ; 40(12): 3821–3827. doi:10.1161/STROKEAHA.109.5603

Trisha M. Kesar, PT, PhD 1,
Ramu Perumal, PhD 2,
Darcy S. Reisman, PT, PhD 1,2,
 AngelaJancosko, PT 2,
Katherine S. Rudolph, PT, PhD 1,2,
Jill S Higginson, PhD 2,3, and
Stuart A.Binder-Macleod, PT, PhD 1,2
1 Department of Physical Therapy, University of Delaware, Newark, DE
2 Graduate Program in Biomechanics and Movement Science, University of Delaware, Newark, DE
3 Department of Mechanical Engineering, University of Delaware, Newark, DE

 Abstract

Background and Purpose—
Functional electrical stimulation (FES) is a popular post-stroke gait rehabilitation intervention. Although stroke causes multi-joint gait deficits, FES is commonly used only for the correction of swing phase foot drop. Ankle plantar flexor muscles play an important role during gait. The aim of the current study is to test the immediate effects of delivering FES to both ankle plantar flexors and dorsiflexors on post-stroke gait.
Methods—
Gait analysis was performed as subjects (N=13) with chronic post-stroke hemiparesis walked at their self-selected walking speeds during walking with and without FES.
Results—
Compared to delivering FES to only the ankle dorsiflexor muscles during the swing phase,delivering FES to both the paretic ankle plantarflexors during terminal stance and dorsiflexors during swing phase provided the advantage of greater swing phase knee flexion, greater ankle plantar flexion angle at toe-off, and greater forward propulsion. Although FES of both the dorsi- and plantar flexor muscles improved swing phase ankle dorsiflexion compared to no FES, the improvement was less than that observed by stimulating the dorsiflexors alone, suggesting the need to further optimize stimulation parameters and timing for the dorsiflexor muscles during gait.
Conclusions—
In contrast to the typical FES approach of only stimulating ankle dorsiflexor muscles during the swing phase, delivering FES to both the plantar- and dorsiflexor muscles can help to correct post-stroke gait deficits at multiple joints (ankle and knee) during both the swing and stance phases of gait. Our study shows the feasibility and advantages of stimulating the ankle plantar flexors during FES for post-stroke gait.
 

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