Deans' stroke musings: Rehabilitation Strategies and Key Related Mechanisms Involved in Stroke Recovery

Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, April 4, 2020

Rehabilitation Strategies and Key Related Mechanisms Involved in Stroke Recovery

Useless. With no references to protocols or results this is just trying to justify the tyranny of low expectations.

Rehabilitation Strategies and Key Related Mechanisms Involved in Stroke Recovery

By Hideki Nakano

Submitted: May 31st 2019Reviewed: January 7th 2020Published: April 1st 2020

DOI: 10.5772/intechopen.91025

Abstract

Poststroke rehabilitation requires a thorough understanding of the neural mechanisms underlying motor function recovery. This chapter outlines these mechanisms and also discusses the corresponding rehabilitation strategies based on the functional characteristics of the brain. The main topics we discuss are as follows: Although ipsilateral brain region activity is inhibited when using the limbs under normal conditions, it is thought (I don't give a shit what you think, WHAT DO YOU KNOW ABOUT STROKE RECOVERY?)that a decrease in this inhibition and the subsequent increased ipsilateral brain area activity post-injury promote recovery in the damaged contralateral neural network. For optimal poststroke motor function recovery, it is important to normalize the resulting imbalance in brain activity. Therefore, increased corticomotor excitation in the injured hemisphere or decreased excitation in the non-injured hemisphere must be promoted. Rehabilitation strategies include reducing non-paretic limb somatosensory input to decrease excitation in the non-injured hemisphere, increasing paretic limb somatosensory input to increase excitation in the injured hemisphere, increasing excitation in the injured hemisphere through movement training of the paretic hand and anesthesia of the paretic upper arm, increasing excitation in the injured hemisphere, or reducing excitation in the non-injured hemisphere. Considering the functional characteristics of the primary motor area, during the early stages after stroke, it is important to increase the somatosensory input to the paralyzed side and combine mental practices using motor imagery.