Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there. Did your doctor somehow miss the recovery protocol that Pedro Bach-y-Rita used to recover fully back in 1958 with only a partial brain?

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, September 26, 2026

Chronic stress linked to heart damage, other risks in new imaging study

 

Don't let your incompetent? doctor stress you out by NOT HAVING EXACT 100% RECOVERY PROTOCOLS! You doctor has known of this fucking failure since medical school and DONE NOTHING! THAT IS PURE INCOMPETENCE! Get that doctor fired!

Chronic stress linked to heart damage, other risks in new imaging study

Chronic stress is associated with an increased risk of heart damage, according to new data published in the European Journal of Preventive Cardiology.[1] The same study linked living a stressful life to a significantly higher risk of experiencing a heart attack or stroke.

Researchers tracked data from more than 480,000 U.K. Biobank participants, using cardiac MRI scans to identify signs of chronic inflammation. The group then used artificial intelligence to learn more about how such inflammation impacts the heart and impacts a person risk of experiencing a major adverse cardiovascular event (MACE).

Overall, the group found that larger amounts of chronic inflammation—a low-level activation of a person’s immune system—were linked to reduced left ventricular volumes and an increased MACE risk. 

“Our study, which is the largest of its kind, suggests that millions of people could be living with hidden inflammation, which is slowly changing their heart and causing long-term damage—increasing the risk of heart attack and stroke,” corresponding author Declan O’Regan, PhD, a researcher with the MRC Laboratory of Medical Sciences and Imperial College London, explained in a statement. “Chronic inflammation is complicated, but we know it’s tied to our health and driven by a range of lifestyle and economic factors—meaning people may be at more risk just because of their surroundings, their economic status, their family’s health and their lifestyle.”

Chronic inflammation has previously been linked to an increased risk of diabetes and certain cancers. O’Regan noted that a large number of things can contribute to chronic inflammation, including everything from smoking and inactivity, to a person’s genetics.

Bryan Williams, chief scientific officer and chief medical officer of the British Heart Foundation, was not involved in this analysis, but he provided his own perspective in the same statement.

“The research provides valuable insights into how our genes and lifestyle factors, such as smoking, poor mental health and socioeconomic status, can converge to trigger inflammation,” he said. “It also identifies several inflammatory proteins that appear to play a key role in the heart changes linked to chronic inflammation, and it is to be hoped that anti-inflammatory medicines could become an important tool in preventing cardiovascular disease.”

Research teams all over the world have been hard working to develop new ways to reduce inflammation as a way to potentially improve cardiovascular outcomes. These findings help make the case that such studies could be potential game-changers in the years ahead.

Click here to read the full analysis.

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