Deans' stroke musings

Changing stroke rehab and research worldwide now.Time is Brain!Just think of all the trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 493 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It's quite disgusting that this information is not available from every stroke association and doctors group.
My back ground story is here:http://oc1dean.blogspot.com/2010/11/my-background-story_8.html

Saturday, February 18, 2017

Laser-based camera improves view of the carotid artery

You'll have to ask your doctor if this is better at detecting possible problems than ultrasound or blood flow velocity in 4D.
https://www.mdlinx.com/internal-medicine/medical-news-article/2017/02/15/carotid-artery-laser-based-camera-scanning/7054087/?
University of Michigan Health System, 02/15/2017
Michigan Medicine researchers employ novel technology to monitor vulnerabilities for cardiovascular events, aid in diagnosis and treatment.
Strokes and heart attacks often strike without warning. But, a unique application of a medical camera could one day help physicians know who is at risk for a cardiovascular event by providing a better view of potential problem areas.

A new paper in the journal Nature Biomedical Engineering reports proof–of–concept results for this new imaging platform for atherosclerosis.

“The camera actually goes inside the vessels,” says first author Luis Savastano, MD, a Michigan Medicine resident neurosurgeon. “We can see with very high resolution the surface of the vessels and any lesions, such as a ruptured plaque, that could cause a stroke. This technology could possibly find the ‘smoking gun’ lesion in patients with strokes of unknown cause, and may even be able to show which silent, but at–risk, plaques may cause a cardiovascular event in the future.”

The scanning fiber endoscope, or SFE, used in the study was invented and developed by co–author and University of Washington mechanical engineering research professor Eric Seibel, PhD. He originally designed it for early cancer detection by clearly imaging cancer cells that are currently invisible with clinical endoscopes.

The Michigan Medicine team used the instrument for a new application: acquiring high–quality images of possible stroke–causing regions of the carotid artery that may not be detected with conventional radiological techniques. The team worked with senior author Thomas Wang, MD, PhD, who is at the forefront of novel imaging platforms. Wang is professor of internal medicine, biomedical engineering and mechanical engineering, and is the H Marvin Pollard Collegiate Professor of Endoscopy Research.

The team generated images of human arteries using the SFE, which illuminates tissues with multiple laser beams, and digitally reconstructs high–definition images to determine the severity of atherosclerosis and other qualities of the vessel wall.

“In addition to discovering the cause of the stroke, the endoscope can also assist neurosurgeons with therapeutic interventions by guiding stent placement, releasing drugs and biomaterials and helping with surgeries,” Seibel says.

In addition, the SFE uses fluorescence indicators to show key biological features associated with increased risk of stroke and heart attacks in the future.

“The ability to identify and monitor the biological markers that render a plaque unstable and at risk for rupture could enable the detection of individuals within high–risk populations who are most likely to suffer from cardiovascular events, and therefore benefit the most from preventive treatment during the asymptomatic stage,” says B. Gregory Thompson, MD, professor of neurosurgery at the University of Michigan Medical School and a senior author on the new paper.

“In addition, plaque–specific data could help physicians modulate treatment intensity of atherosclerosis, which is currently based on systemic surrogates such us cholesterol and blood sugar levels and occurrence of cardiovascular events such as stroke or myocardial infarction.”

All research is in the pre–clinical phase.
Go to Abstract Print Article Summary Cat 2 CME Report

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