Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, July 1, 2021

Cerebral hyperperfusion syndrome after mechanical thrombectomy

Is your doctor prepared for treating Cerebral hyperperfusion syndrome if needed?

 Cerebral hyperperfusion syndrome after mechanical thrombectomy

  1. Markus Kneihsl1,
  2. Christian Enzinger1,2,
  3. Thomas Gattringer1,2
  1. Correspondence to Dr Markus Kneihsl, Department of Neurology, Medical University of Graz, Graz 8036, Austria; markus.kneihsl@medunigraz.at

Case summary

A male patient in his late 50s who had untreated atrial fibrillation received mechanical thrombectomy for right middle cerebral artery occlusion (MCA), with complete recanalization. After initial neurological improvement, he developed headache and altered vigilance 2 days post-thrombectomy. Transcranial duplex sonography (TCD) showed increased blood flow velocities in the entire ipsilateral (recanalized) MCA-M1 segment.1 2 Brain MRI with angiography excluded focal stenosis and showed vasogenic edema, hemorrhagic transformation of the ischemic infarct, and cerebral hyperperfusion in the right MCA territory (figure 1). Because of this finding, the patient underwent intense blood pressure (BP) control (systolic BP target …

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Footnotes

  • Contributors MK: case report design, acquisition and interpretation of data, manuscript preparation. CE: critical revision of the manuscript content. TG: case report concept and design, interpretation of data, manuscript preparation, critical revision of the manuscript content.

  • Funding The authors have not declared a specific grant for this research from any funding agency in the public, commercial or not-for-profit sectors.

  • Competing interests None declared.

  • Provenance and peer review Not commissioned; externally peer reviewed.

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