Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, December 7, 2022

Chapter: Making Memories Last; How sleep promotes neuroplasticity By Randolph F. Helfrich, Robert T. Knight

You can't expect your doctor to read this and create protocols for sleeping, and you can't do this on your own since that would be practicing medicine without a license. You're screwed.

ByRandolph F. Helfrich, Robert T. Knight


Making Memories Last

Making Memories Last

How sleep promotes neuroplasticity
ByRandolph F. Helfrich, Robert T. Knight
Edition 1st Edition
First Published 2022
Imprint Routledge
Pages 17
eBook ISBN 9780429342356
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ABSTRACT

Decades of research have demonstrated that sleep benefits memory formation and restores cognitive resources. While the behavioral benefits of sleep are well established, the neurophysiological underpinnings are less clear. In particular, it remains unknown how memories are transferred from short- to long-term storage. While initial theories were largely centered on rapid eye movement (REM) sleep, several contemporary theories converged on the notion that non-REM (NREM) sleep is actively engaged in memory consolidation. NREM sleep is dominated by prominent neuronal oscillations, such as cortical slow waves (<1.25 Hz), thalamo-cortical sleep spindles (12–16 Hz), and hippocampal ripple oscillations (80–200 Hz). Here we provide an overview of how selective synchronization of neuronal oscillations promotes information reactivation, transfer, and consolidation during sleep. We explore the neocortical-hippocampal dialogue in support of information selection and distribution, and we discuss the concept of cross-frequency coupling as a neural mechanism of information transfer. In particular, we focus on how time-varying, oscillatory activity can promote a neurophysiological milieu that mediates neuroplasticity. Taken together, we will review evidence of how sleep provides optimal conditions for neuroplasticity and outline that disruption of sleep can contribute to age- and disease-related memory impairments and cognitive decline.


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