Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Monday, November 7, 2011

Potential New Treatment For Stroke

Another hyperacute research possibility.
http://www.medicalnewstoday.com/releases/235406.php
Even though stroke is the third leading cause of death in the United States, there is only one approved treatment. Furthermore, fewer than 5% of stroke patients benefit from this treatment. New therapeutic targets are therefore urgently needed.

Katrin Andreasson and colleagues, at Stanford University School of Medicine, Stanford, have now identified the protein EP4 as a potential new target for the treatment of stroke. Key to their suggestion that EP4 could provide therapeutic benefit to stroke patients, was the observation that therapeutic systemic administration of a selective EP4 agonist after stroke reduced brain damage and long-term behavioral deficits in mice.

TITLE: Signaling via the prostaglandin E2 receptor EP4 exerts neuronal and vascular protection in a mouse model of cerebral ischemia

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