Deans' stroke musings

Changing stroke rehab and research worldwide now.Time is Brain!Just think of all the trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 493 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It's quite disgusting that this information is not available from every stroke association and doctors group.
My back ground story is here:http://oc1dean.blogspot.com/2010/11/my-background-story_8.html

Sunday, January 14, 2018

Contributions of voluntary activation deficits to hand weakness after stroke

You are stating the obvious, have you no clue what the definition of a stroke is? My god, I can't activate my finger flexors or extensors because that area is dead.
http://search.naric.com/research/rehab/redesign_record.cfm?search=2&type=all&criteria=J77371&phrase=no&rec=135309&article_source=Rehab&international=0&international_language=&international_location=
Topics in Stroke Rehabilitation , Volume 23(6) , Pgs. 384-392.

NARIC Accession Number: J77371.  What's this?
ISSN: 1074-9357.
Author(s): Hoffman, Gilles; Conrad, Megan O.; Qiu, Dan; Kamper, Derek G..
Publication Year: 2016.
Number of Pages: 9.
Abstract: Study investigated the role of diminished capacity to voluntarily active finger flexor and extensor muscles as one possible neurological mechanism for hand weakness in hemiparetic stroke survivors. Two groups of stroke survivors, 9 with ‘‘severe’’ and 9 with ‘‘moderate’’ hand impairment, and one group of 9 neurologically intact individuals (controls) participated. Subjects were asked to create isometric flexion force and extension force, respectively, with the tip of the middle finger. The maximum voluntary force (MVF) and the maximum stimulated force (MSF) produced by an applied train of electrical current pulses (MSF) were recorded for flexion and extension. Percent voluntary activation (PVA) was computed from MVF and MSF. Significant deficits in both MVF and PVA were observed for stroke subjects compared to control subjects. For example, activation deficits were greater than 80 percent for extensor digitorum communis (EDC) for the ‘‘severe’’ group. Maximum voluntary force and PVA deficits were greater for EDC than for flexor digitorum superficialis for stroke subjects with severe impairment. Maximum voluntary force and PVA correlated significantly for stroke subjects but not for control subjects.The findings indicate that although extrinsic finger muscles could be successfully recruited electrically, voluntary excitation of these muscles was substantially limited in stroke survivors. Thus, finger weakness after stroke results predominantly from the inability to fully activate the muscle voluntarily.
Descriptor Terms: DEXTERITY, HEMIPLEGIA, MUSCULAR IMPAIRMENTS, STROKE.


Can this document be ordered through NARIC's document delivery service*?: Y.

Citation: Hoffman, Gilles, Conrad, Megan O., Qiu, Dan, Kamper, Derek G.. (2016). Contributions of voluntary activation deficits to hand weakness after stroke.  Topics in Stroke Rehabilitation , 23(6), Pgs. 384-392. Retrieved 1/14/2018, from REHABDATA database.

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