Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, December 7, 2013

Hypertension: The Link Between Erectile Dysfunction and Coronary Artery Disease

I just liked this because my solution to this problem is beer.
Follow the thought process;
endothelial function was significantly improved only after beer consumption
Ergo, beer will solve this. You can thank me after seeing what your doctors' pontification is.

http://online.liebertpub.com/doi/full/10.1089/jomh.2013.1503
Jacob Rajfer, MD,1 and Martin M. Miner, MD2,3
1Department of Urology, UCLA School of Medicine, Los Angeles, California.
2Department of Family Medicine and Department of Urology, Warren Alpert School of Medicine, Brown University, Providence, Rhode Island.
3Men's Health Center, The Miriam Hospital, Providence, Rhode Island.
Address correspondence to:
Martin M. Miner, MD
The Warren Alpert School of Medicine of Brown University
164 Summit Avenue
Providence, RI 02906
E-mail:

ABSTRACT

The ability to attain and then maintain an erection is the result of a dynamic balance between arterial inflow and cavernosal outflow. Any imbalance that either decreases arterial inflow and/or increases cavernosal outflow may result in symptomatic erectile dysfunction (ED). Current data suggest that vasculogenic ED is of arterial origin and primarily the result of the development of systemic endothelial dysfunction (EnD). Since systemic EnD is heralded as an early marker of cardiovascular disease, specifically coronary artery atherosclerotic heart disease (CAD), it has been suggested that the onset of ED can be used as a marker for either the presence, or future development, of CAD. However, the failure to find arterial dysfunction in some young men with largely vasculogenic ED, combined with the recognition that most men with ED, regardless of age, appear to have at least some cavernosal smooth muscle dysfunction, has prompted a reexamination of the roles of vascular endothelium and the cavernosal smooth muscle in the development of aging-related ED, as well as a reexamination of the significance of ED as a marker of subsequent cardiovascular disease. The unifying concept that explains all these events is the aging-related apoptosis that occurs within vascular smooth muscle. In the penis, this smooth muscle apoptosis will cause cavernosal veno-occlusive dysfunction (CVOD), which initially affects a man's refractory period. In the peripheral vascular system, the apoptotic process leads to an increase in peripheral vascular resistance, and patients may present with hypertension. Hypertension and CVOD, then, represent the same pathophysiologic disorder in two different organ systems.

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