Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, June 8, 2017

Abstracts for the 24th Annual Meeting of the American Society for Neural Therapy and Repair

I'm sure these 30 pages of abstracts have multiple trials needing followup. What EXACTLY is your doctor and stroke hospital doing to facilitate that? Or are they both just HAVING THEIR HEADS UP THEIR FUCKING ASSES DOING NOTHING? Suggested followup bolded in red.
I'm looking forward to which is better caffeinol or nanocoffee?
http://www.ingentaconnect.com/contentone/cog/ct/2017/00000026/A00104s1/art00001?crawler=true&mimetype=application/pdf 
Presidential Symposium: How Efficient Is Clinical Translation in Neurology?

Modulating the Inflammation-Associated Stroke Vasculome With Human Endothelial Progenitor Cells

Higher Cognitive Abilities Prior to Stroke Influence Neuroplasticity and Mobilization of Human Bone Marrow Stem Cells in Affording Neuroprotection

Mesenchymal Stem Cell-Derived Exosomes Mediate Neurovascular Protection

Proof of Concept—CRISPR-Cas9 Lipid Nanoparticles as an Efficient Delivery Tool for Cultured Cells and in Animal Models

KDEL Receptors—Novel ER Stress Response Genes

Age-Dependent Remodeling and Gene Expression Changes of the Cerebral Pial Collaterals Following Traumatic Brain Injury

The Tricyclic Antidepressant Medication Nortriptyline Inhibits -Synuclein Accumulation, Aggregation, and Toxicity in Multiple In Vitro and In Vivo Models

LRRK2 Pathways: Relevance for Inherited and Sporadic Parkinson’s Disease

Mitochondrial Unfolded Protein Response (mtUPR) Dysfunction During the Progression of Alzheimer’s Disease

Complementation of Lmx1a/Pitx3 -Null Embryos With Porcine Blastomeres Generates Chimeric Fetal Porcine Brain

Nanocoffee Displays Neuroprotective Effects in Traumatic Brain Injury Models

Allele-Specific Silencing of Mutant Huntingtin Gene Following Injection of Complexed Lipid Nanoparticle and Transcription Activator-Like Effectors In Vivo

Targeted Gene Regulation in Genetically Linked Neurological Disorders Using CRISPR/Cas9 and Transcription Activator-Like Effectors

Human Cord Blood Plasma as a Potential Therapeutic for ALS

Potential Repair of the Blood–Spinal Cord Barrier Coincides With Reduction of Microhemorrhages in the Spinal Cord of Symptomatic ALS Mice After Intravenous Human Bone Marrow CD34 + Cell Transplantation

Liposomes as Ang-(1–7) Carriers to the CNS for Ischemic Stroke Therapy

Inhibition of Drp1 Mitochondrial Translocation Provides Neural Protection in Dopaminergic System in a Parkinson’s Disease Model Induced by MPTP

Targets for Global Brain Delivery of Recombinant Protein

Histological Correlates of Traumatic Brain Injury-Induced Anxiety Behaviors: A Target for Hyperbaric Oxygen Therapy

ECM Hydrogel Injection for the Treatment of Stroke

Improving Viability of hNSCs by Optimizing Biomechanical Stresses During Cell Injection

The Synaptic Scaffolding Protein Homer1c Is Necessary for Successful Learning and Memory and for Induction of Group 1 Metabotropic Glutamate Receptor-Mediated Long-Term Depression

Nonhuman Primate Modeling of Zika Virus Vertical Transmission

Phase 1/2a Study to Evaluate the Safety of Neural Stem Cells in Patients With Parkinson’s Disease
Investigating a Role for Connexin 43 in Hippocampal Neurogenesis Following Moderate Traumatic Brain Injury 

Astaxanthin Modulates Cognitive Function in Young and Aged Mice

Hyperbaric Oxygen Therapy Effects on Traumatic Brain Injury- Associated Behavioral Impairments

LISPRO, an Ionic Cocrystal of Lithium, Mitigates Alzheimer-Like Pathological Changes in the Mice

Interrogating the Role of Peripheral-Derived Hematopoietic Cells in Tissue Homeostasis Following Brain Trauma

Cerebral Aneurysm Healing: MCP-1–IL-6–OPN Pathway

Proteomic Comparison of Aged and Young Microglia Highlights Biological Consequences of Aging

Modulation of Microglia Cannabinoid Receptor 2 to Ameliorate Neuroinflammation in Parkinson’s Disease

A Nonhuman Primate Model Facilitates the Development of Neuroplasticity-Related Treatment Targets for Anxiety and Depressive Disorders

The Role of Maternal Gut Microbiome in Perinatal Neurodevelopment: Implications for Neurodevelopmental Disorders

Absence of Direct Cell-to-Cell Contact in Human iPSC-Induced Neuroprotection Against Stroke In Vitro Reveals Novel Stem Cell Anti-Inflammatory Secreted Factors and Filopodia Extension

Neural Stem Cell-Encoded Hypoxia Inducible Factor-1a (HIF-1a) Is Essential for the Endogenous Regenerative Response to Ischemic Injury in a Mouse Model of Stroke

Postinjury Therapeutic Effects of 3,6 -Dithio Pomalidomide on Traumatic Brain Injury 


Zika Virus Dysregulates Gene Expression and Alters Protein Secretion in Neural Stem Cells

Priming Effects of Endotoxemia on Neutrophil Activation, Neuroinflammation, and Reperfusion Injury Following Transient Global Ischemia

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