Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Monday, August 6, 2018

Teetotalism in midlife makes you more likely to develop dementia? What the research actually says

Your doctor will never tell you of any benefits of alcohol. You can't listen to me since I have no medical training. You are on your own for your health, better start studying up.

Don't do this, I am a complete outlier and should never be followed.

My listing of positives; don't follow me. 

I actually consider alcohol to be the great social engagement lubricant, and stroke survivors needs lots of social interaction to prevent dementia. 


Alcohol for these 12 reasons.



A little daily alcohol may cut stroke risk




Six healthy reasons to drink more beer   Red wine benefits are in this one also.



10 Health Benefits of Whiskey

Study: For those over 90, alcohol better than exercise for longevity

 

NIH withdrawal from controversial trial leaves the question: Does alcohol prevent CVD?

You are completely on your own figuring out what to do about alcohol.

Your choice on what to do with this, your doctor will disapprove. 

The Common Drink Linked To High Intelligence

The latest here:

Teetotalism in midlife makes you more likely to develop dementia? What the research actually says

Imagine if someone told you that not drinking alcohol makes you more likely to develop dementia. That's certainly a bold statement to make, but it is one that some media reports have put forward, leaving the public wondering whether taking up drinking alcohol will somehow be better for their brains.


A recent article in the British Medical Journal has rekindled the scientific argument over the relationship between abstaining from and developing dementia. The study involved 9,000 civil servants working in London, all of whom were aged between 35 and 55 when the study began between 1985 and 1988. When the study ended in 1993, their average age was 50. The participants' medical records were analysed to identify diagnoses of dementia—more than 23 years, on average, after the study had finished. The results found that abstinence in was associated with a 45% higher risk of developing dementia, compared with people who consumed between one and 14 units of alcohol per week.

The findings for those who drank above the recommended 14 units a week were perhaps less surprising. For every seven unit per week increase above 14 units there was a significant 17% increase in the risk of dementia. The study concluded: "These results suggest that abstention and are associated with an increased risk of dementia, although the underlying mechanisms are likely to be different in the two groups."

A confounded nuisance

So what are we to make of this research? In studies such as this, one of the many pitfalls of exploring the association between an exposure variable (alcohol) and the outcome variable (dementia) is the presence of confounders. Confounders are a statistician's worse nightmare – they are variables that may justifiably be associated with both the exposure and outcome variable.

In this study, abstainers with higher levels of illnesses such as heart disease, diabetes and obesity were those at highest risk of dementia. It may be that having a physical illness is the reason that these people abstained from alcohol. These same physical illnesses may also be the reason for developing dementia. In this way, physical illness unrelated to alcohol consumption could be a confounder for both abstinence and dementia. Drinking may just be a red herring.

The other big problem with studies that explore how teetotalism and dementia are connected is the age range covered. The study started looking at habits at a minimum age of 35 and finished when the average age was 50. But what about people who drank heavily in their younger years or above the age of 55? This is particularly important, as it is likely that a large proportion of the study group will have been part of the "baby boomer" generation. This generation has seen the highest rise in rates of alcohol misuse over the past 15 years – a period of time which is well outside the study's range.

We should also be aware that between one-quarter and two-thirds of older people start drinking more than the recommended 14 unit limit for the first time after the age of 60. These drinking patterns at ages before and after the study period could well have influenced the results. Those identified as teetotal during the study period may well have started drinking later in life

Another spirit level

More than 50 years ago, the late Griffiths Edwards, a professor of addictions psychiatry stated: "It would be too optimistic to suppose that the relative under-representation of subjects in the older age groups … is just explained by older people having generally got the treatment they required or having reverted to normal drinking … it seems likely that this finding is in part a hint of the diminished life expectancy of the alcoholic."

Humans have a longer lifespan these days, so there are more people living into their 70s and beyond – which has also provided the opportunity to look at alcohol and dementia in more detail. By examining the differences between alcohol-related dementia and more common forms, such as Alzheimer's Disease, we begin to see that they are different in many ways. For example, the problems with word-finding seen in Alzheimer's Disease is rarely seen in alcohol-related dementia, which also presents much earlier with changes in behaviour.

Researchers need to be sure that they are defining alcohol-related dementia more accurately for these kinds of studies to be definitive. If not, it is highly likely that they are looking at other dementias with other risk factors, such as high blood pressure or diabetes.

Alcohol is a drug that harms increasing numbers of – and we need to get the facts right about how it affects the brain. Maybe we can't handle the truth about drinking. All it takes is the mere flicker of hope as to the benefits of being teetotal to fan the flames of misinformation.

For those that do drink alcohol, people drinking above lower risk limits has, once again, been given a salutary warning. It is one that we should continue to heed – but we also need to get the message out there that alcohol-related needs to be rediscovered and investigated again.


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