Deans' stroke musings: New biomarker for acute ischaemic stroke: Plasma glycogen phosphorylase isoenzyme BB

Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Monday, October 23, 2017

New biomarker for acute ischaemic stroke: Plasma glycogen phosphorylase isoenzyme BB

No clue what this means.
https://www.mdlinx.com/internal-medicine/medical-news-article/2017/10/18/acute-ischaemic-stroke-plasma-levels-of/7475775/?

Journal of Neurology, Neurosurgery & Psychiatry | October 18, 2017

Park KY, et al. - The target herein was to investigate the rise in plasma levels of glycogen phosphorylase BB (GPBB) isoform, among patients with acute ischaemic stroke (AIS). A robust response was illustrated by GPBB, with regard to acute ischaemia and high sensitivity for small infarcts. GPBB could be beneficial as a stand-alone marker for acute brain ischaemia if the results were affirmed in more diverse populations including stroke mimics.