Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, October 19, 2017

Alzheimer’s Linked To Too Much of This In Your Diet - Sugar

Your doctor hasn't before and never will come up with a diet protocol, so you are completely on your own. Danger, danger Will Robinson.
Alzheimer’s Linked To Too Much of This In Your Diet - Sugar

The researchers studied samples of brain tissue from people with and without Alzheimer’s disease. Excess sugar in the diet could play an important role in the development of Alzheimer’s disease, new research finds. Too much glucose (sugar) in the diet damages a vital enzyme which helps fight the early stages of Alzheimer’s disease. Dr Omar Kassaar, the study’s first author, said: “Excess sugar is well known to be bad for us when it comes to diabetes and obesity, but this potential link with Alzheimer’s disease is yet another reason that we should be controlling our sugar intake in our diets.” The researchers studied samples of brain tissue from people with and without Alzheimer’s disease. They found that sugar can damage an enzyme called MIF (macrophage migration inhibitory factor). Professor Jean van den Elsen, a study co-author, explained:> “We’ve shown that this enzyme is already modified by glucose in the brains of individuals at the early stages of Alzheimer’s disease. We are now investigating if we can detect similar changes in blood. Normally MIF would be part of the immune response to the build-up of abnormal proteins in the brain, and we think that because sugar damage reduces some MIF functions and completely inhibits others that this could be a tipping point that allows Alzheimer’s to develop. MIF helps to fight the build up of abnormal proteins in the brain, which are characteristic of Alzheimer’s. The reduction of MIF activity by glucose could eventually lead to a ‘tipping point’ in Alzheimer’s progression. Dr Rob Williams, a study co-author, said: “Knowing this will be vital to developing a chronology of how Alzheimer’s progresses and we hope will help us identify those at risk of Alzheimer’s and lead to new treatments or ways to prevent the disease. The study was published in the journal Scientific Reports (Kassaar et al., 2017).

2 comments:

  1. If looks as though they have the hypothetical cause, but what about effect? Can't there be a study correlating having type 2 diabetes and Alzheimer's to test the hypothesis? Or looking at long-term BS data (a1c) w it? It looks easy to me.

    ReplyDelete
    Replies
    1. Of course it looks easy to solve, we understand cause and effect. Researchers don't really care about cause and effect, just where the next research money is coming in. And leaving these open questions can lead to the next research project.

      Delete