Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, October 21, 2017

Substance in coffee delays onset of diabetes in laboratory mice

With the possible connection that Alzheimers is linked to diabetes, your stroke hospital should have a 24 hour coffee station, and you should be non-catheterized so you can practice walking to the bathroon and all the ADLs associated with that. I have 150 posts on coffee.   so no reason for your doctor and stroke hospital not to know of the benefits.  Unless they are so fucking lazy that they are

WAITING FOR SOMEONE ELSE TO SOLVE THE PROBLEM?

Scientists are finding more evidence that could link Type 2 diabetes with Alzheimer's disease

Substance in coffee delays onset of diabetes in laboratory mice

American Chemical Society News | September 22, 2017
In recent years, researchers have identified substances in coffee that could help quash the risk of developing type 2 diabetes. But few of these have been tested in animals. Now in study appeared in the Journal of Natural Products, scientists report that one of these previously untested compounds appears to improve cell function and insulin sensitivity in laboratory mice. The finding could spur the development of new drugs to treat or even prevent the disease.

Some studies suggest that drinking three to four cups of coffee a day can reduce the risk of developing type 2 diabetes. Initially, scientists suspected that caffeine was responsible for this effect. But later findings discounted this possibility, suggesting that other substances in coffee may have a more important role. In a previous laboratory study, Fredrik Brustad Mellbye, Søren Gregersen and colleagues found that a compound in coffee called cafestol increased insulin secretion in pancreatic cells when they were exposed to glucose. Cafestol also increased glucose uptake in muscle cells just as effectively as a commonly prescribed antidiabetic drug. In this new study, the researchers wanted to see if cafestol would help prevent or delay the onset of type 2 diabetes in mice.

The researchers divided mice that are prone to develop type 2 diabetes into three groups. Two of the groups were fed differing doses of cafestol. After 10 weeks, both sets of cafestol-fed mice had lower blood glucose levels and improved insulin secretory capacity compared to a control group, which was not given the compound. Cafestol also didn’t result in hypoglycemia, or low blood sugar, a possible side effect of some antidiabetic medications. The researchers conclude that daily consumption of cafestol can delay the onset of type 2 diabetes in these mice, and that it is a good candidate for drug development to treat or prevent the disease in humans.

No comments:

Post a Comment