Do you really think your competent? doctor will have the nutritionist validate that all hospital meals do not have this and will create EXACT DIET PROTOCOLS for all these needs?
For dementia prevention; for cognitive improvement; for cholesterol reduction; for plaque removal; for Parkinsons prevention; for inflammation reduction; etc.
Do you prefer your doctor and hospital incompetence NOT KNOWING? OR NOT DOING?
Your doctors have known of this problem for a year and a half. How fucking incompetent can they be and still be employed by your hospital?
Zero-calorie sweetener linked to heart attack and stroke, study finds
February 2023
Your competent? doctor also needs to ensure further human testing occurs!
This is why your nutritionist needs to validate amounts in your diet protocol:
What food has erythritol in it?
It's also found in mushrooms and fermented foods like beer, cheese, sake, soy sauce and wine. In addition to whole foods, erythritol is commercially produced for use in baked goods, beverages, candies, chewing gums, chocolates and tabletop sweetener packets.
The latest here:
Ingestion of the Non-Nutritive Sweetener Erythritol, but Not Glucose, Enhances Platelet Reactivity and Thrombosis Potential in Healthy Volunteers
Arteriosclerosis, Thrombosis, and Vascular Biology
Abstract
BACKGROUND:
Although
artificial and non-nutritive sweeteners are widely used and generally
recognized as safe by the US and European Union regulatory agencies,
there have been no clinical trials to assess either long-term
cardiovascular disease risks or short-term cardiovascular
disease–relevant phenotypes. Recent studies report that fasting plasma
levels of erythritol, a commonly used sweetener, are clinically
associated with heightened incident cardiovascular disease risks and
enhance thrombosis potential in vitro and in animal models. Effects of
dietary erythritol on thrombosis phenotypes in humans have not been
examined.
METHODS:
Using
a prospective interventional study design, we tested the impact of
erythritol or glucose consumption on multiple indices of
stimulus-dependent platelet responsiveness in healthy volunteers (n=10
per group). Erythritol plasma levels were quantified with liquid
chromatography tandem mass spectrometry. Platelet function at baseline
and following erythritol or glucose ingestion was assessed via both
aggregometry and analysis of granule markers released.
RESULTS:
Dietary
erythritol (30 g), but not glucose (30 g), lead to a >1000-fold
increase in erythritol plasma concentration (6480 [5930–7300] versus
3.75 [3.35–3.87] μmol/L; P<0.0001) and exhibited acute
enhancement of stimulus-dependent aggregation responses in all subjects,
agonists, and doses examined. Erythritol ingestion also enhanced
stimulus-dependent release of the platelet dense granule marker
serotonin (P<0.0001 for TRAP6 [thrombin activator peptide 6] and P=0.004 for ADP) and the platelet α-granule marker CXCL4 (C-X-C motif ligand-4; P<0.0001 for TRAP6 and P=0.06
for ADP). In contrast, glucose ingestion triggered no significant
increases in stimulus-dependent release of either serotonin or CXCL4.
CONCLUSIONS:
Ingestion
of a typical quantity of the non-nutritive sweetener erythritol, but
not glucose, enhances platelet reactivity in healthy volunteers, raising
concerns that erythritol consumption may enhance thrombosis potential.
Combined with recent large-scale clinical observational studies and
mechanistic cell-based and animal model studies, the present findings
suggest that discussion of whether erythritol should be reevaluated as a
food additive with the Generally Recognized as Safe designation is
warranted.
REGISTRATION:
URL: https://www.clinicaltrials.gov; Unique identifier: NCT04731363.
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