Deans' stroke musings

Changing stroke rehab and research worldwide now.Time is Brain!Just think of all the trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 493 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It's quite disgusting that this information is not available from every stroke association and doctors group.
My back ground story is here:http://oc1dean.blogspot.com/2010/11/my-background-story_8.html

Thursday, May 10, 2018

Cholesterol in the blood could cause dementia

Except that this dire warning doesn't include the information this earlier reference did. That cholesterol does not cross the blood-brain barrier and other information that 25% of the cholesterol in your body is in your brain. What does your doctor have to say about these competing ideas?

Your Brain Needs Cholesterol - David Perlmutter M.D.

Cholesterol could cause dementia, research warns.

Scientists found a link between the fatty substance in the blood and ­amyloid-beta protein — a key player in the onset of Alzheimer’s disease.

They discovered that cholesterol in the brain is a trigger for the formation of clusters of amyloid-beta. They eventually lead to the formation of amyloid plaques in a toxic chain reaction that results in the death of brain cells.

An international team led by Cambridge University researchers said the identification of a new “pathway” in the brain where amyloid-beta sticks together, or aggregates, could represent a fresh target for potential treatments.



“We’re not saying that cholesterol is the only trigger for the aggregation process, but it’s certainly one of them.”

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