Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, December 30, 2014

Glutamate Excitotoxicity and Neurodegeneration

Your doctor should be able to use this understanding to figure out if two of the processes involved in the neuronal cascade of death are the reason for your 33% dementia chance post-stroke from an Australian study?
What exactly is your doctor doing to stop those two neuronal death processes and maybe prevent your dementia?
http://scholar.google.com/scholar_url?url=http://omicsonline.com/open-access/glutamate-excitotoxicity-and-neurodegeneration-1747-0862-1000141.pdf&hl=en&sa=X&scisig=AAGBfm1cEQLfAP2l2iqHDqrC0PO2p08zoA&oi=scholaralrt
Ezza HSA1* and Khadrawyb YA2
1Zoology Department, Faculty of Science, Cairo University, Giza, Egypt
2Medical Physiology Department, Medical Division, National Research Center, Giza, Egypt
*Corresponding author: Ezza HSA, Zoology Department, Faculty of Science, Cairo University, Giza, Egypt, Tel: 20237753565; Fax: +202 33387758; E-mail:
hebasal@yahoo.com
Rec Date: May 20, 2014; Acc date: October 06, 2014; Pub date: October 08, 2014
Copyright: © 2014 Ezza HSA. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use,
distribution, and reproduction in any medium, provided the original author and source are credited.

Abstract
Glutamate plays crucial roles in the physiology of the central nervous system as it can control many functions such as memory, learning, cognitive, emotional, endocrine and other visceral functions. In addition, glutamate is the major excitatory neurotransmitter in the mammalian central nervous system. It has the potential to be involved in the pathogenesis of many CNS diseases either due to excessive release, reduced uptake or alteration of receptor functions. Growing evidence links glutamate excitotoxicity to various neurodegenerative diseases as cerebral ischemia, epilepsy, Alzheimer's disease, Parkinsons' disease and multiple sclerosis. In addition, several
environmental pollutants result in excessive glutamatergic neurotransmission and may eventually lead to neurodegenerative diseases.
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