Deans' stroke musings

Changing stroke rehab and research worldwide now.Time is Brain!Just think of all the trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 493 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It's quite disgusting that this information is not available from every stroke association and doctors group.
My back ground story is here:

Saturday, October 8, 2016

Epigenetic clock predicts life expectancy, UCLA-led study shows

What is your blood age? Highly unlikely you can get your doctor to test this on you and get your insurance to pay for it. If my age was early, I would eat and drink unhealthily and get the bucket list done now.
University of California - Los Angeles Health News, 09/30/2016
New research finds 5 percent of population ages faster, faces shorter lifespan.
Why do some people lead a perfectly healthy lifestyle yet still die young? A new international study suggests that the answer lies in our DNA. UCLA geneticist Steve Horvath led a team of 65 scientists in seven countries to record age–related changes to human DNA, calculate the biological age of blood and estimate a person’s lifespan. A higher biological age — regardless of chronological age — consistently predicted an earlier death. The findings are published in the journal Aging. “Our research reveals valuable clues into what causes human aging, marking a first step toward developing targeted methods to slow the process,” said Horvath, the principal investigator and a professor of human genetics and biostatistics at the David Geffen School of Medicine at UCLA and Fielding School of Public Health. Drawing on 13 sets of data, including the landmark Framingham Heart Study and Women’s Health Initiative, a consortium of 25 institutions analyzed the DNA in blood samples collected from more than 13,000 people in the United States and Europe. Applying a variety of molecular methods, including an epigenetic clock developed by Horvath in 2013, the scientists measured the aging rates of each individual. The clock calculates the aging of blood and other tissues by tracking methylation, a natural process that chemically alters DNA over time. By comparing chronological age to the blood’s biological age, the scientists used the clock to predict each person’s life expectancy. The preliminary finding may explain why some individuals die young — even when they follow a nutritious diet, exercise regularly, drink in moderation and don’t smoke.
Go to Abstract Print Article Summary Cat 2 CME Report

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