Deans' stroke musings

Changing stroke rehab and research worldwide now.Time is Brain!Just think of all the trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 493 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It's quite disgusting that this information is not available from every stroke association and doctors group.
My back ground story is here:

Wednesday, April 19, 2017

Insomnia symptoms appear to increase risk for MI, stroke

So what is your doctors' sleep protocol so you get enough rest and aren't fatigued all the time?
3 years ago this was already identified and I bet your doctor did fucking nothing.

Insomnia may significantly increase stroke risk  April, 2014 

The latest here, bet your doctor still does nothing:
Patients with insomnia symptoms have an increased risk for future cardio-cerebral vascular events, including MI and stroke, according to findings published in the European Journal of Preventive Cardiology.
Sleep disorders are common in the general population, and sleep health should be included in clinical risk assessment,” Qiao He, a Master’s degree student at China Medical University in Taichung, Taiwan, said in a press release. “Health education is needed to increase public awareness of insomnia symptoms and the potential risks so that people with sleep problems are encouraged to seek help.”
Researchers reviewed data from 15 studies of 23 cohorts (n = 160,867) whose participants were older than 18 years, had at least one self-reported symptom of insomnia at baseline and had outcomes that included incidence or death from cardio-cerebral vascular events such as acute MI, CHD, stroke, HF or combined events. The symptoms of insomnia that researchers focused on were difficulty initiating sleep, difficulty maintaining sleep, early morning awakening and nonrestorative sleep.
Participants who reported difficulty initiating sleep experienced a link to cardio-cerebral vascular events (RR = 1.27; 95% CI, 1.15-1.4) with significant heterogeneity (I2 = 54.9%; P =.001). Using subgroup analysis, researchers found that the risk for cardio-cerebral vascular events in participants from non-European countries was “borderline significant” vs. 1.25 in participants from European countries. Women who reported difficulty initiating sleep had a slightly higher risk for events (RR = 1.36; 95% CI, 1.1-1.67) compared with men (RR = 1.31; 95% CI, 1.18-1.45).
Those who reported difficulty maintaining sleep had an increased risk for cardio-cerebral vascular events compared with those without the symptom (RR = 1.11; 95% CI, 1.04-1.19) with minor heterogeneity (I2 = 14.4%; P = .278). The subgroup analysis for this symptom of insomnia had similar results to those who reported difficulty initiating sleep, in which women had a slightly higher risk.
Early morning awakening was marginally associated with the risk for cardio-cerebral vascular events (RR = 1.02; 95% CI, 0.95-1.1) with no heterogeneity (I2 = 0%; P = .458).
Participants with nonrestorative sleep saw a higher risk for cardio-cerebral vascular events (RR = 1.18; 95% CI, 1.05-1.33) with mild heterogeneity (I2 = 38.5%; P =.123). The risk in women who did not feel rested after sleep was higher by 11% (RR = 1.28; 95% CI, 1.1–1.48) compared with men with the same symptoms (RR = 1.17; 95% CI, 0.99-1.38).
“It is well known that there are biological differences in genetic and sexual hormones and sex differences in stress and reactions to stress as well, which in turn cause women to be more prone to insomnia, as has been indicated in several studies,” He and colleagues wrote. “However, we cannot absolutely conclude that insomnia is more dangerous for women.” – by Darlene Dobkowski

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